Author

G. E. Kim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Hiniker· 1 shared B. J. Backes· 1 shared D. J. Maly· 1 shared D. W. Dawson· 1 shared F. R. Papa· 1 shared H. Htwe Htwe· 1 shared H. R. Shin· 1 shared J. A. Yano· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Hiniker

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Author profile →

2 papers

Recent·Most cited

Lysosomal retargeting of Myoferlin mitigates membrane stress to enable pancreatic cancer growth

S. Gupta, J. A. Yano, H. Htwe Htwe, H. R. Shin, Z. Cakir, T. Ituarte, K. W. Wen, G. E. Kim, R. Zoncu, D. W. Dawson, R. M. Perera

This study identifies Myoferlin as a lysosomal membrane integrity factor in pancreatic cancer, showing its role in lysosome health and tumor growth.

Nature Cell Biology · Mar 8, 2021Peer-reviewedCancer Biology

Parallel signaling through IRE1α and PERK regulates pancreatic neuroendocrine tumor growth and survival

P. C. Moore, J. Y. Qi, M. Thamsen, R. Ghosh, J. Peng, M. J. Gliedt, R. Meza-Acevedo, R. E. Warren, A. Hiniker, G. E. Kim, D. J. Maly, B. J. Backes, F. R. Papa, S. A. Oakes

This study shows that targeting the UPR components IRE1α and PERK can inhibit pancreatic neuroendocrine tumor growth by inducing apoptosis through reciprocal signaling.

bioRxiv · Sep 18, 2019Cancer Biology