Author

A. Hiniker

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Christensen· 1 shared A. Haghani· 1 shared A. Nguyen· 1 shared A. Unapanta· 1 shared B. J. Backes· 1 shared B. Li· 1 shared C. Balen· 1 shared C. Finch· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperEvolutionary Biology

A. Christensen

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Author profile →

3 papers

Recent·Most cited

ApoE isoforms establish distinct antioxidant trajectories across the aging brain

S. J. Shemtov, M. Cacciottolo, A. Christensen, B. Li, A. Haghani, M. C. Rice, H. Anson, C. S. Chung, W. Cohn, H. Zhang, W. J. Mack, A. Hiniker, C. Lee, T. E. Morgan, E. Head, , Max A. Thorwald

This study shows ApoE isoforms influence antioxidant pathways differently across aging, with ApoE4 showing stronger transcriptional responses but less effective protein production, linking to neurodegenerative risk.

bioRxiv · Nov 9, 2026 · PreprintEvolutionary Biology

Endogenous Rab38 regulates LRRK2's membrane recruitment and substrate Rab phosphorylation in melanocytes

A. Unapanta, F. Shavarebi, J. Porath, C. Balen, A. Nguyen, J. Tseng, Y. Shen, M. Liu, P. Lis, S. Di Pietro, A. Hiniker

This study identifies Rab38 as a physiological regulator of LRRK2 function in melanocytes, showing it enhances LRRK2 membrane recruitment and substrate phosphorylation.

bioRxiv · Jun 20, 2022Cell Biology

Parallel signaling through IRE1α and PERK regulates pancreatic neuroendocrine tumor growth and survival

P. C. Moore, J. Y. Qi, M. Thamsen, R. Ghosh, J. Peng, M. J. Gliedt, R. Meza-Acevedo, R. E. Warren, A. Hiniker, G. E. Kim, D. J. Maly, B. J. Backes, F. R. Papa, S. A. Oakes

This study shows that targeting the UPR components IRE1α and PERK can inhibit pancreatic neuroendocrine tumor growth by inducing apoptosis through reciprocal signaling.

bioRxiv · Sep 18, 2019Cancer Biology