Author

G. Vassiliou

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:CITE-seq· 1 Assay:DNA methylation· 1

Frequent Co-Authors

B. J. P. Huntly· 2 shared A. Damaskou· 1 shared A. de Bruin· 1 shared A. E. Tijhuis· 1 shared A. Mupo· 1 shared A. S. Bach· 1 shared A. Strong· 1 shared A. van den Brink· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

B. J. P. Huntly

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3 papers

Recent·Most cited

HOXA9 forms a repressive complex with nuclear matrix-associated protein SAFB to maintain acute myeloid leukemia

S. Agrawal Singh, J. Bagri, G. Giotopoulos, D. Azazi, S. J. Horton, J. W. Houghton, S. Anand, A. S. Bach, F. Stedham, R. Antrobus, G. Vassiliou, D. Sasca, H. Yun, A. Whetton, B. J. P. Huntly

This study identifies a repressive HOXA9-SAFB complex in AML that maintains disease by inhibiting differentiation and apoptosis, suggesting therapeutic targets.

bioRxiv · Oct 12, 2022 · PreprintCancer Biology

Cancer tolerance to chromosomal instability is driven by Stat1 inactivation in vivo

M. Schubert, C. Hong, L. J. Jilderda, M. Requesens-Rueda, A. E. Tijhuis, J. E. Simon, P. L. Bakker, J. Cooper, A. Damaskou, R. Wardenaar, B. Bakker, S. Gupta, A. van den Brink, L. Andrade-Ruiz, M. H. Koster, , F. Foijer

The paper identifies Stat1 inactivation as a mechanism by which aneuploid cancers evade immune surveillance in mice and humans.

bioRxiv · Dec 3, 2021 · PreprintCancer Biology

Genetic modification of primary human B cells generates translationally-relevant models of high-grade lymphoma

R. Caeser, M. Di Re, J. A. Krupka, J. Gao, M. Lara-Chica, J. M. L. Dias, S. L. Cooke, R. Fenner, Z. Usheva, H. Runge, P. A. Beer, H. Eldaly, H.-K. Pak, C.-S. Park, G. Vassiliou, , D. J. Hodson

This study generates synthetic lymphoma models by genetically modifying primary human B cells to investigate oncogenic mutations in DLBCL.

bioRxiv · Apr 26, 2019 · PreprintCancer Biology