Author

J. C. Mar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

E. J. Wolvetang· 2 shared A. J. van Waardenberg· 1 shared A. Laslett· 1 shared A. T. Fard· 1 shared C. Nefzger· 1 shared E. W. Tate· 1 shared G. Balistreri· 1 shared G. H. Talbo· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

E. J. Wolvetang

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inhibition of host N-myristoylation compromises the infectivity of SARS-CoV-2 due to Golgi-bypassing egress from lysosomes and endoplasmic reticulum

S. H. Saber, M. R. Shaker, J. Sng, N. Yak, S. D. Morrison, S. Pars, H. Zheng, G. Pietrogrande, T. Binder, S. Lu, M. Floetenmeyer, R. Ojha, T. Quirin, J. Ruuska, T. Smura, , M. Joensuu

This study shows that inhibiting host N-myristoylation reduces SARS-CoV-2 infectivity by impairing viral envelope protein maturation and causing Golgi-bypassing egress.

bioRxiv · Mar 3, 2023 · PreprintMicrobiology

Substrate stiffness facilitates improved induced pluripotent stem cell production through modulation of both early and late phases of cell reprogramming

M. M. Chowdhury, S. Samuel Zimmerman, H. Leeson, C. Nefzger, J. C. Mar, A. Laslett, J. M. Polo, E. J. Wolvetang, J. J. Cooper-White

This study shows that medium stiffness substrates enhance iPSC production by improving both early and late reprogramming phases, leading to higher numbers and better quality iPSCs.

bioRxiv · Feb 27, 2023 · PreprintBioengineering

Redox signaling regulates breast cancer metastasis via HIF1alpha-stimulated EMT dynamics and metabolic reprogramming

Z. Ren, M. Dharmaratne, H. Liang, O. Benard, M. Morales-Gallego, K. Suyama, A. T. Fard, J. C. Mar, M. Prystowsky, L. Norton, R. B. Hazan

This study shows that GPx2 loss in mammary tumors enhances metastasis through HIF1-driven EMT and metabolic changes, identified via scRNA-seq.

bioRxiv · Aug 29, 2022 · PreprintCancer Biology