Author

J. Woods

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Assay:proteomics· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1

Frequent Co-Authors

A. Cupp· 1 shared A. Flora· 1 shared B. Johannesson· 1 shared B. Liau· 1 shared C. Cordes· 1 shared D. Bhattacharya· 1 shared D. D. Hwang· 1 shared E. Kozera· 1 shared

Activity by Year

2018-2023 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 6 mutual co-author links

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1 shared paperCell Biology

A. Cupp

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Engineering Human Pluripotent Stem Cell Lines to Evade Xenogeneic Transplantation Barriers

H. A. Pizzato, P. Alonso-Guallart, J. Woods, B. Johannesson, J. P. Connelly, T. A. Fehniger, J. Atkinson, S. M. Pruett-Miller, F. J. Monsma, D. Bhattacharya

This study genetically modifies human pluripotent stem cells to evade immune rejection in mice by knocking out HLA genes and adding inhibitory factors, leading to persistent teratomas in immunocompetent models.

bioRxiv · Jun 27, 2023Immunology

Base Editor Scanning Reveals Activating Mutations of DNMT3A

E. M. Garcia, N. Z. Lue, J. K. Liang, W. K. Lieberman, D. D. Hwang, J. Woods, B. Liau

The paper uses base editor mutagenesis to identify activating DNMT3A mutations, revealing hyperactivating variants in the ADD domain that remain functional in the presence of a heterozygous R882H mutation.

bioRxiv · Apr 12, 2023Biochemistry

Functional genomics identifies extension of complex N-glycans as a mechanism to evade lysis by natural killer cells

X. Zhuang, J. Woods, Y. Ji, S. Scheich, F. Mo, M. Voss, H. Urlaub, K.-T. Pan, E. O. Long

The paper identifies SPPL3 as a gene that enables tumor cells to evade NK cell lysis by altering N-glycan structures, and shows that inhibiting N-glycan maturation restores NK sensitivity.

bioRxiv · Apr 3, 2023 · PreprintImmunology

Human Dermal Fibroblast Subpopulations and Epithelial Mesenchymal Transition Signals in Hidradenitis Suppurativa Tunnels are Normalized by Spleen Tyrosine Kinase Antagonism in Vivo

A. Flora, R. Jepsen, E. Kozera, J. Woods, G. Cains, M. Razieta, S. Jensen, M. Malone, J. W. Frew

The paper identifies fibroblast subpopulations and EMT signals in HS tissue and shows that Spleen Tyrosine Kinase inhibition reduces inflammation and fibroblast activity.

bioRxiv · Feb 23, 2023Immunology

Trafficking of Cholesterol from Lipid Droplets to Mitochondria in Bovine Luteal Cells: Acute Control of Progesterone Synthesis

M. R. Plewes, C. Cordes, E. Przgrodzka, H. Talbott, J. Woods, A. Cupp, J. Davis

This study shows LH and PKA activate HSL phosphorylation, enabling cholesterol release from lipid droplets to mitochondria for progesterone synthesis in bovine luteal cells.

bioRxiv · Sep 5, 2018Cell Biology