Author

Katrina Savory

King's College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 2

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Approach:experimental assay· 2 Organism:human· 1

Frequent Co-Authors

Aina Mogas BarconsiD· 1 shared Ana Belén Malpartida· 1 shared Anna Lavayssiere· 1 shared Anshua GhoshiD· 1 shared Benedikt Mathias KessleriD· 1 shared Brent James RyaniD· 1 shared Clive G. BallardiD· 1 shared Dag AarslandiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Aina Mogas BarconsORCID

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2 papers

Recent·Most cited

Synaptotoxic forms of amyloid-b and a-synuclein act through a common pathway.

Elena M. Ribé, Anshua Ghosh, Wojciech Pasławski, Katrina Savory, Clive G. Ballard, P. Morin, Diana Cash, Frank Hirth, Gareth Haydn Williams, Per Svenningsson, Dag Aarsland, Richard C. M. Siow, Richard Killick

The paper shows that synaptotoxic forms of amyloid-beta and alpha-synuclein act through the same Wnt/PCP pathway to cause dendritic spine loss in neurodegenerative diseases.

bioRxiv · Feb 13, 2025 · PreprintNeuroscience

USP30 inhibition improves mitochondrial health through both PINK1-dependent and independent mechanisms

Matthew G Williamson, Rachel Heon‐Roberts, Sarah N. J. Franks, Elliot D. Mock, Hannah B. L. Jones, Elena Britti, Ana Belén Malpartida, Mahmoud Adel Bassal, Martha Lavelle, Jack P. Connor, Aina Mogas Barcons, Katherine L. Hammond, Katrina Savory, Pavandeep Rai, Anna Lavayssiere, , Brent James Ryan

This study shows USP30 inhibition improves mitochondrial health through both PINK1-dependent and independent mechanisms, enhancing mitophagy and mitochondrial efficiency in neurons.

bioRxiv · Feb 3, 2025 · PreprintNeuroscience