Author

M. J. Vliem

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:proteomics· 1

Frequent Co-Authors

M. Gloerich· 2 shared A. A. Khalil· 1 shared A. Krotenberg-Garcia· 1 shared C. Perez-Gonzalez· 1 shared D. Matic Vignjevic· 1 shared E. A. Strating· 1 shared H. Canever· 1 shared H. J. G. Snippert· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

M. Gloerich

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Mechanical regulation of cell fate transitions underlying colorectal cancer metastasis formation

M. C. van der Net, M. J. Vliem, L. J. S. Kemp, C. Perez-Gonzalez, E. A. Strating, A. Krotenberg-Garcia, R. M. Houtekamer, K. B. van den Anker, J. L. Monster, H. J. G. Snippert, A. A. Khalil, J. van Rheenen, S. J. Suijkerbuijk, O. Kranenburg, D. Matic Vignjevic, M. Gloerich

This study shows that mechanical signals from the Collagen I-rich matrix drive fate transitions in colorectal cancer cells, enabling metastasis despite lacking Lgr5 expression.

bioRxiv · Sep 17, 2023 · PreprintCancer Biology

A mechanical G2 checkpoint controls epithelial cell division through E-cadherin-mediated regulation of Wee1-Cdk1

L. Donker, M. J. Vliem, H. Canever, M. Gomez-Gonzalez, M. Bosch-Padros, W.-J. Pannekoek, X. Trepat, N. Borghi, M. Gloerich

The paper shows epithelial cells use a mechanical G2 checkpoint regulated by E-cadherin adhesions to control division in response to cell density changes.

bioRxiv · Nov 29, 2021Cell Biology