M. Mandal

BRWD1 establishes epigenetic states for germinal center initiation, maintenance, and function

This study shows BRWD1 is essential for maintaining epigenetic states in germinal center B cells, affecting their function and subset identity.

BRWD1 orchestrates chromatin topology by converting static to dynamic cohesin complexes

The paper shows BRWD1 converts static to dynamic cohesin complexes in pre-B cells, enabling chromatin topology changes needed for cell fate transitions.

STAT3 signaling in B cells controls germinal center zone organization and recycling

This study shows STAT3 signaling in B cells controls germinal center zone organization and recycling, affecting long-lived plasma cell and memory B cell output.

Microliter spotting and micro-colony observation: a rapid and simple approach for counting bacterial colony-forming units

This paper introduces a rapid method for counting bacterial colony-forming units using micro-colony observation, achieving results comparable to traditional dilution plating with reduced time and labor.

Size dependent regeneration capacity of functionalized Capra ear derived micro-tissue scaffolds for treatment of cartilage defects

This study evaluates the regeneration capacity of size-dependent micro-tissue scaffolds derived from Capra ear cartilage for cartilage defect treatment in rabbits.

Proton egress pathway during the S1 to S2 transition of the Oxygen Evolving Complex ofPhotosystem II

The paper uses computational methods to analyze proton transfer pathways during the S1 to S2 transition of the Oxygen Evolving Complex in Photosystem II, identifying the broad channel as the most likely proton exit route.

Optical Coherence Tomography Reveals Self-Organizing Di-Fork Architecture of Mice Cutaneous Scars

This paper uses SS-OCT to visualize the self-organizing di-fork architecture of mouse cutaneous scars and links it to mechanobiological processes.

B-1a cells acquire their unique characteristics by bypassing the pre-BCR selection stage

This study shows that B-1a cells bypass the pre-BCR selection stage by developing in fetal liver with reduced IL-7R/STAT5 levels, leading to self-reactive BCRs.