Author

M. Poddar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Research Focus:cancer & tumor microenvironment· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Bell· 4 shared S. Hu· 4 shared S. Singh· 4 shared A. Singhi· 3 shared J. Tao· 3 shared K. Nejak-Bowen· 3 shared S. ko· 3 shared S. Liu· 3 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersPathology

A. Bell

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4 papers

Recent·Most cited

Dynamic control of metabolic zonation and liver repair by endothelial cell Wnt2 and Wnt9b revealed by single cell spatial transcriptomics using Molecular Cartography

S. Hu, S. Liu, B. Yu, M. Poddar, S. Singh, C. Cao, J. McGaughey, A. Bell, L. L. Blazer, J. Adams, S. Sidhu, S. Angers, S. P. Monga

This study identifies endothelial cell Wnt2 and Wnt9b as key regulators of liver zonation and repair, showing their therapeutic potential in acute liver failure using single-cell spatial transcriptomics.

bioRxiv · Mar 18, 2022Pathology

β-Catenin-NFκB-CFTR interactions in cholangiocytes regulate inflammation and fibrosis during ductular reaction

S. Hu, J. Russell, S. Liu, R. Rai, K. Kosar, J. Tao, E. Hurley, M. Poddar, S. Singh, A. Bell, D. Shin, R. Raeman, A. Singhi, K. Nejak-Bowen, S. ko, S. P Monga

This study identifies a β-catenin-NFκB-CFTR interactome in cholangiocytes that regulates inflammation and fibrosis during ductular reaction in liver disease.

eLife · Oct 5, 2021Peer-reviewedPathology

NOTCH-YAP1/TEAD-DNMT1 axis regulates hepatocytereprogramming into intrahepatic cholangiocarcinoma

S. Hu, L. M. Molina, J. Tao, S. Liu, M. Hassan, S. Singh, M. Poddar, A. Bell, D. Sia, M. Oertel, R. Raeman, K. Nejak-Bowen, A. Singhi, J. Luo, S. P. Monga, S. Ko

The paper identifies a NOTCH-YAP1/TEAD-DNMT1 axis that drives hepatocyte reprogramming into intrahepatic cholangiocarcinoma and reveals its role in tumor development.

bioRxiv · Dec 3, 2020 · PreprintCancer Biology

Compensatory hepatic adaptation accompanies permanent absence of intrahepatic biliary network due to YAP1 loss in liver progenitors

L. M. Molina, J. Zhu, Q. Li, T. Pradhan-Sundd, K. Sayed, N. Jenkins, R. Vats, S. Ko, S. Hu, M. Poddar, S. Singh, J. Tao, P. Sundd, A. Singhi, S. C. Watkins, , S. P. Monga

This study shows YAP1 is essential for bile duct development and reveals hepatocyte adaptation in its absence.

bioRxiv · Oct 21, 2020Pathology