Author

M. V. Gonzalez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

J. P. Garifallou· 2 shared A. Boehm· 1 shared A. De Martin· 1 shared A. Ferreira· 1 shared A. G. Freud· 1 shared A. Rostami· 1 shared B. Austin· 1 shared B. Ciric· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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2 shared papersImmunology

J. P. Garifallou

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Author profile →

4 papers

Recent·Most cited

Inhibition of NFAT promotes loss of tissue resident uterine natural killer cells and attendant pregnancy complications in humans

R. Asiimwe, B. Knott, M. E. Greene, E. Wright, M. Bell, D. Epstein, S. D. Yates, M. V. Gonzalez, S. Fry, E. Boydston, S. Clevenger, J. E. Locke, B. E. Brocato, C. M. Burgan, R. Burney, , P. M. Porrett

The paper identifies NFAT inhibition as a cause of reduced uterine natural killer (uNK) cells and pregnancy complications in humans through analysis of uterus transplant recipients.

bioRxiv · Mar 7, 2024 · PreprintImmunology

A novel cryopreservation and biobanking strategy to study lymphoid tissue stromal cells in human disease

J. D. Brandstadter, A. De Martin, M. Lutge, A. Ferreira, B. T. Gaudette, Y. Stanossek, S. Wang, M. V. Gonzalez, E. Camiolo, G. Wertheim, B. Austin, D. Allman, M. S. Lim, D. C. Fajgenbaum, J. C. Aster, , I. Maillard

This study demonstrates that cryopreservation of lymphoid tissue allows for the study of non-hematopoietic stromal cells in human disease, preserving cell type proportions and transcriptional profiles.

bioRxiv · Feb 6, 2023Immunology

CSF-1 maintains pathogenic but not homeostatic myeloid cells in the central nervous system during autoimmune neuroinflammation.

D. Hwang, L. L. W. Ishiwaka, A. Boehm, Z. Sahin, G. Casella, S. Jang, M. Seyedsadr, M. V. Gonzalez, J. P. Garifallou, H. Hakonarson, G.-X. Zhang, A. Rostami, B. Ciric

This study shows that blocking CSF-1 reduces harmful myeloid cells in EAE without affecting beneficial ones, suggesting CSF-1 as a therapeutic target for MS.

bioRxiv · Oct 23, 2020Immunology

Neuroinflammation and EIF2 signaling persist in an HiPSC tri-culture model of HIV infection despite antiretroviral treatment

S. K. Ryan, M. V. Gonzalez, J. P. Garifallou, F. C. Bennett, K. S. Williams, H. K. Hakonarson, S. A. Anderson, K. L. Jordan-Sciutto

This study uses a HiPSC tri-culture model to show that HIV infection and antiretroviral treatment persistently affect neuroinflammation and EIF2 signaling in the CNS.

bioRxiv · Sep 24, 2019Pathology