Author

P. A. Wark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:human· 4 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1

Frequent Co-Authors

P. M. Hansbro· 4 shared J. C. Horvat· 3 shared P. S. Pathinayake· 3 shared A. T. Reid· 2 shared K. F. Budden· 2 shared P. C. Veerati· 2 shared A. C.-Y. Hsu· 1 shared A. F. Faiz· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersCell Biology

P. M. Hansbro

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Fecal microbial transfer and complex carbohydrates mediate protection against COPD

K. F. Budden, S. Shukla, K. L. Bowerman, S. L. Gellatly, D. L. A. Wood, N. Lachner, S. Idrees, V. Patel, A. F. Faiz, S. F. Rehman, C. Donovan, C. Alemao, S. Shen, K. S. Vanka, J. Mason, , P. M. Hansbro

This study shows that fecal microbial transfer and complex carbohydrates can reduce COPD symptoms in mice and humans, linking gut microbiota to disease progression.

bioRxiv · Oct 16, 2023 · PreprintPhysiology

Comparison of commercially available differentiation media on morphology, function, and virus-host interaction in conditionally reprogrammed human bronchial epithelial cells

N. T. Awatade, A. T. Reid, K. S. Nichol, K. F. Budden, P. C. Veerati, P. S. Pathinayake, C. L. Grainge, P. M. Hansbro, P. A. Wark

This study compares two differentiation media for conditionally reprogrammed human bronchial epithelial cells, finding structural and functional differences but no impact on viral infection outcomes.

bioRxiv · Apr 12, 2023 · PreprintCell Biology

Single cell RNA-seq identifies inflammation-induced loss of CFTR-expressing airway ionocytes in non-eosinophilic asthma

L. Chen, G. A. Hoefel, P. S. Pathinayake, A. Reid, C. Kelly, T. HuiYing, R. Y. Kim, P. M. Hansbro, S. L. Brody, P. S. Foster, J. C. Horvat, C. Riveros, P. A. Wark, G. E. Kaiko

This study identifies inflammation-induced loss of CFTR-expressing ionocytes in non-eosinophilic asthma using single-cell RNA-seq and confirms it in human and murine models.

bioRxiv · Apr 26, 2022 · PreprintCell Biology

SARS-CoV-2 Spike protein promotes hyper-inflammatory response that can be ameliorated by Spike-antagonistic peptide and FDA-approved ER stress and MAP kinase inhibitors in vitro

A. C.-Y. Hsu, G. Wang, A. T. Reid, P. C. Veerati, P. S. Pathinayake, K. Daly, J. R. Mayall, P. M. Hansbro, J. C. Horvat, F. Wang, P. A. Wark

This study shows that SARS-CoV-2 spike protein induces inflammation and epithelial damage, which can be reduced by an antagonistic peptide and FDA-approved drugs.

bioRxiv · Sep 30, 2020 · PreprintImmunology