Author

P. O. Ljungdahl

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4 Cell Biology· 2

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Research Focus:host-pathogen & immune response· 5 Organism:C. elegans· 4 Approach:experimental assay· 3 Approach:genetic perturbation / CRISPR· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:Drosophila· 1 Assay:imaging· 1

Frequent Co-Authors

F. G. S. Silao· 4 shared K. Ryman· 4 shared M. Ward· 3 shared C. Chen· 2 shared N.-N. Liu· 2 shared T. Jiang· 2 shared A. Besold· 1 shared A. Broggi· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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4 shared papersMicrobiology

F. G. S. Silao

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

Diverse mechanisms control amino acid-dependent environmental alkalization by Candida albicans

F. G. S. Silao, V. D. Valeriano, E. Uddström, E. Falconer, P. O. Ljungdahl

This study identifies Gdh2 and its regulatory mechanisms in Candida albicans for amino acid-dependent environmental alkalization, showing its role in virulence and microbial community dynamics.

bioRxiv · Aug 4, 2023 · PreprintCell Biology

Proline catabolism is key to facilitating Candida albicans pathogenicity

F. G. S. Silao, T. Jiang, B. Bereczky-Veress, A. Kuehbacher, K. Ryman, N. Uwamohoro, S. Jenull, F. Nogueira, M. Ward, T. Lion, C. F. Urban, S. Rupp, K. Kuchler, C. Chen, C. Peuckert, P. O. Ljungdahl

This study shows that proline catabolism is essential for Candida albicans pathogenicity, as mutations in proline utilization genes reduce virulence in animal models.

bioRxiv · Jan 17, 2023 · PreprintMicrobiology

The ER membrane chaperone Shr3 acts in a progressive manner to assist the folding of related plasma membrane transport proteins

I. Myronidi, A. Ring, P. O. Ljungdahl

This study shows Shr3, an ER chaperone, interacts with AAP transporters progressively during their translation, preventing non-productive interactions.

bioRxiv · Jul 5, 2020 · PreprintCell Biology

Glutamate dehydrogenase (Gdh2)-dependent alkalization is dispensable for escape from macrophages and virulence of Candida albicans

F. G. S. Silao, K. Ryman, T. Jiang, M. Ward, N. Hansmann, N.-N. Liu, C. Chen, P. O. Ljungdahl

This study shows that Gdh2-dependent alkalization is not required for C. albicans to escape macrophages or maintain virulence, challenging the existing model of amino acid catabolism in phagosomes.

bioRxiv · Jan 20, 2020Microbiology

Mitochondrial proline catabolism activates Ras1/cAMP/PKA-induced filamentation in Candida albicans

F. G. S. Silao, M. Ward, K. Ryman, A. Wallstrom, B. Brindefalk, K. Udekwu, P. O. Ljungdahl

This study shows that mitochondrial proline metabolism via PUT1 and PUT2 enhances ATP production, activates the Ras1/cAMP/PKA pathway, and promotes filamentation in C. albicans, contributing to virulence.

bioRxiv · Jul 23, 2018Microbiology

Intersection of phosphate transport, oxidative stress and TOR signalling in Candida albicans virulence

N.-N. Liu, P. Uppuluri, A. Broggi, A. Besold, K. Ryman, H. Kambara, N. Solis, V. Lorenz, W. Qi, M. Acosta Zaldivar, S. N. Emami, B. Bao, D. An, F. Bonilla, M. Sola-Visner, , J. R. Koehler

This study shows that Pho84 in Candida albicans affects virulence by modulating oxidative stress through Sod3 and TORC1 pathways.

bioRxiv · May 11, 2018Microbiology