Author

Rabia Khan

Cornell UniversityPrimary ORCID profile (0000-0003-1434-2020 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Rabia KhaniD1 paper · University of Oslo

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

Bobak ParangiD· 2 shared Eric E. GardneriD· 2 shared John BlenisiD· 2 shared Michal J. NagieciD· 2 shared Alexandra FrancianiD· 1 shared Anders Peter MutveiiD· 1 shared Andre Chavez· 1 shared Ashish SaxenaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCancer Biology

Bobak ParangORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identification of Adjuvants that Enhance the Magnitude and Durability of Antibody Responses to a Nanoparticle-Based Malaria Vaccine

Yogesh Nepal, Alexandra Francian, Yevel Flores-Garcia, Bryce T. Roberts, Rabia Khan, Garrett Wondra, Aurélie Demierre, Falko Apel, Fidel Zavala, Bryce Chackerian

bioRxiv · Oct 14, 2026 · PreprintImmunology

Propionate metabolism dysregulation promotes drug-tolerant persister cell survival in non-small cell lung cancer

Bobak Parang, Liron Yoffe, Rabia Khan, Zhongchi Li, Michal J. Nagiec, Eric E. Gardner, Yiwey Shieh, Rachel S. Heise, Ashish Saxena, Nasser Khaled Altorki, John Blenis

This study shows that propionate metabolism dysregulation, through MMA accumulation and EMT activation, promotes drug-tolerant persister cell survival in NSCLC.

bioRxiv · Oct 15, 2025 · PreprintCancer Biology

Lysosome-dependent nutrient scavenging underliesstress adaptation during epithelial-to-mesenchymaltransition

Michal J. Nagiec, Paola M. Cavaliere, Friederike Dündar, Derrick M. Spencer, Nikos Koundouros, Joana B. Nunes, Sejeong Shin, Sang-Oh Yoon, Julie Han, Andre Chavez, Olivia M. Kester, Rabia Khan, Paul Zumbo, Eric E. Gardner, Bobak Parang, , John Blenis

This study shows that invasive cancer cells use lysosome-mediated nutrient scavenging during EMT, bypassing plasma membrane transport, and that this process is regulated by c-MYC downregulation.

bioRxiv · Sep 22, 2025 · PreprintCell Biology