Author

S. A. Singh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

C. L. Clift· 2 shared E. Aikawa· 2 shared M. Aikawa· 2 shared M. C. Blaser· 2 shared T. Pham· 2 shared A. C. Sonawane· 1 shared A. Daugherty· 1 shared C. Ching· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBioengineering

C. L. Clift

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Intracellular Proteomics and Extracellular Vesiculomics as a Metric of Disease Recapitulation in 3D Bioprinted Aortic Valve Arrays

C. L. Clift, M. C. Blaser, W. Gerrits, M. E. Turner, A. C. Sonawane, T. Pham, J. L. Andresen, O. S. Fenton, J. M. Grolman, F. Buffolo, F. J. Schoen, J. Hjortnaes, J. D. Muehlschlegel, D. D. Mooney, M. Aikawa, , E. Aikawa

This study compares 3D-bioprinted and 2D models of calcific aortic valve disease using proteomics and vesiculomics to assess disease recapitulation.

bioRxiv · Jun 22, 2023Bioengineering

Endothelial cells secrete small extracellular vesicles bidirectionally containing distinct cargo to uniquely reprogram vascular cells in the circulation and vessel wall

S. Raju, S. R. Botts, M. C. Blaser, K. Prajapati, T. W. W. Ho, C. Ching, N. J. Galant, L. Fiddes, R. Wu, C. L. Clift, T. Pham, W. L. Lee, S. A. Singh, E. Aikawa, J. E. Fish, K. L. Howe

This study shows endothelial cells release distinct extracellular vesicles to reprogram monocytes and smooth muscle cells in circulation and vessel wall.

bioRxiv · Apr 28, 2023 · PreprintCell Biology

Second Heart Field-derived Cells Contribute to Angiotensin II-mediated Ascending Aortopathies

H. Sawada, Y. Katsumata, H. Higashi, C. Zhang, Y. Li, S. Morgan, L. H. Lee, S. A. Singh, J. Z. Chen, D. A. Howatt, J. J. Moorleghen, M. K. Franklin, D. L. Rateri, Y. H. Shen, S. A. LeMaire, , A. Daugherty

This study shows SHF-derived cells contribute to AngII-induced ascending aortic pathologies through LRP1 and TGF-β signaling, increasing PAI1 expression.

bioRxiv · Feb 2, 2020Pathology