Author

S. Ferri-Borgogno

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cancer & tumor microenvironment· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

A. Maitra· 3 shared S. Gupta· 3 shared B. Ghosh· 2 shared J. Zhao· 2 shared A. Hosein· 1 shared A. M. McGee· 1 shared A. Singhi· 1 shared A. Verma· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersCancer Biology

A. Maitra

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Author profile →

4 papers

Recent·Most cited

Long-term ovarian cancer survivors: spatial transcriptomics depict ligand-receptor crosstalk heterogeneity at the tumor-stroma interface

S. Ferri-Borgogno, Y. Zhu, J. Sheng, J. Burks, J. Gomez, K. K. Wong, S. T. Wong, S. Mok

bioRxiv · Jun 4, 2022Cancer Biology

Oncogenic KRAS Requires Complete Loss of BAP1 Function for Development of Murine Intrahepatic Cholangiocarcinoma

R. K. Marcus, S. Ferri-Borgogno, A. Hosein, W. C. Foo, B. Ghosh, J. Zhao, K. Rajapakshe, J. Brugarolas, A. Maitra, S. Gupta

This study develops a mouse model showing that complete loss of BAP1 function is required for KRAS-driven intrahepatic cholangiocarcinoma.

bioRxiv · Oct 12, 2021Cell Biology

Paradoxical role of AT-rich interactive domain 1A in restraining pancreatic carcinogenesis

S. Ferri-Borgogno, S. Barui, A. M. McGee, T. Griffiths, P. K. Singh, C. G. Piett, B. Ghosh, S. Bhattacharyya, A. Singhi, K. Pradhan, A. Verma, Z. Nagel, A. Maitra, S. Gupta

This study investigates the role of Arid1a in pancreatic carcinogenesis using mouse models and cell lines, finding that its loss impairs tumor growth and DNA repair but allows for later aggressive cancer development.

bioRxiv · Nov 8, 2019Cancer Biology

YAP1 Oncogene is a Context-specific Driver for Pancreatic Ductal Adenocarcinoma

B. Tu, J. Yao, S. Ferri-Borgogno, J. Zhao, S. Chen, Q. Wang, L. Yan, X. Zhou, C. Zhu, S. Bang, Q. Chang, C. A. Bristow, Y. Kang, H. Zheng, H. Wang, , H. Ying

This study identifies YAP1 as a context-specific driver for the squamous subtype of pancreatic ductal adenocarcinoma and shows WNT5A promotes tumor progression through YAP1 activation.

bioRxiv · Aug 17, 2019Cancer Biology