S. M. Abel

Propulsive cell entry diverts pathogens from immune degradation by remodeling the phagocytic synapse

The paper shows that pathogens can avoid immune degradation by using physical forces during entry, not just virulence factors.

Particle deformability enables control of interactions between membrane-anchored nanoparticles

This study shows that deformable membrane-anchored nanoparticles can be controlled through their mechanical properties to modulate interactions and self-assembly.

Exchange between compartments regulates steady states and stochastic switching of a multisite phosphorylation network

This study shows that protein exchange between compartments affects the steady states and stochastic switching in a two-site phosphorylation network, with switching rates peaking at intermediate exchange rates.

Topographical analysis of immune cell interactions reveals a biomechanical signature for immune cytolysis

The paper analyzes immune cell interactions using traction force microscopy to identify biomechanical features associated with cytotoxic T cell activity.

Organization and Dynamics of Crosslinked Actin Filaments in Confined Environments

This study uses simulations to show how crosslinker binding and physical confinement affect actin filament organization and dynamics in confined spaces.

Membrane-mediated interactions between hinge-like particles

This study investigates how hinge-like particles adsorbed on an elastic membrane interact, showing that adhesion strength and hinge stiffness control their aggregation behavior.

Adsorption of semiflexible polymers in crowded environments

This study uses simulations to show that crowding increases adsorption of semiflexible polymers on repulsive surfaces, with effects dependent on polymer stiffness, crowder size, and volume fraction.

Mechanical feedback enables catch bonds to selectively stabilize scanning microvilli at T-cell surfaces

This study shows that catch bonds selectively stabilize scanning microvilli on T cells by slowing their motion and increasing receptor engagement time.

The Cdc42 GEF, Gef1, promotes uniform protein distribution along the actomyosin ring to enable concentric furrowing

The paper shows that Gef1 and endocytic events ensure uniform Cdc15 distribution along the actomyosin ring, enabling concentric furrow formation during cytokinesis in fission yeast.