Author

T. Shoji

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1 Pharmacology & Toxicology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1

Frequent Co-Authors

C. Sumi· 2 shared K. Hirota· 2 shared M. Kusunoki· 2 shared T. Uba· 2 shared Y. Matsuo· 2 shared A. Okamoto· 1 shared H. Bono· 1 shared H. Tanaka· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCancer Biology

C. Sumi

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A jasmonate-responsive ERF transcription factor regulates steroidal glycoalkaloid biosynthesis genes in eggplant

T. Shoji, K. Saito

This study shows that overexpressing SmJRE4 in eggplant increases SGA biosynthesis genes, and jasmonate induces SmJRE4 while ethylene reduces this effect.

bioRxiv · Nov 6, 2022Plant Biology

Methylation of 23S rRNA G748 and the ribosomal protein L22 Lys-94 are critical factors for maintaining the association between ribosome stalling and proteome composition in Streptococcus pneumoniae

T. Shoji

This study identifies m1G748 methylation and L22 K94 as critical factors linking ribosome stalling and proteome composition in S. pneumoniae.

bioRxiv · Apr 14, 2021Microbiology

Cancerous phenotypes associated with hypoxia-inducible factors are not influenced by the volatile anesthetic isoflurane in renal cell carcinoma

C. Sumi, Y. Matsuo, M. Kusunoki, T. Shoji, T. Uba, T. Iwai, H. Bono, K. Hirota

This study examines whether isoflurane affects cancer cell phenotypes linked to hypoxia-inducible factors in renal cell carcinoma, finding no positive influence of isoflurane on these traits.

bioRxiv · Feb 26, 2019Cancer Biology

Propofol induces metabolic reprogramming and cell death in a mitochondrial electron transport chain-dependent manner

C. Sumi, A. Okamoto, H. Tanaka, K. Nishi, M. Kusunoki, T. Shoji, T. Uba, Y. Matsuo, T. Adachi, J.-I. Hayashi, K. Takenaga, K. Hirota

This study shows propofol causes metabolic reprogramming and cell death by impairing mitochondrial function, specifically complexes I and III, and this effect is exacerbated by preexisting mitochondrial dysfunction.

bioRxiv · Nov 16, 2017Pharmacology & Toxicology