Author

T. Uba

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Sumi· 2 shared K. Hirota· 2 shared M. Kusunoki· 2 shared T. Shoji· 2 shared Y. Matsuo· 2 shared A. Okamoto· 1 shared H. Bono· 1 shared H. Tanaka· 1 shared

Activity by Year

2017-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCancer Biology

C. Sumi

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2 papers

Recent·Most cited

Cancerous phenotypes associated with hypoxia-inducible factors are not influenced by the volatile anesthetic isoflurane in renal cell carcinoma

C. Sumi, Y. Matsuo, M. Kusunoki, T. Shoji, T. Uba, T. Iwai, H. Bono, K. Hirota

This study examines whether isoflurane affects cancer cell phenotypes linked to hypoxia-inducible factors in renal cell carcinoma, finding no positive influence of isoflurane on these traits.

bioRxiv · Feb 26, 2019Cancer Biology

Propofol induces metabolic reprogramming and cell death in a mitochondrial electron transport chain-dependent manner

C. Sumi, A. Okamoto, H. Tanaka, K. Nishi, M. Kusunoki, T. Shoji, T. Uba, Y. Matsuo, T. Adachi, J.-I. Hayashi, K. Takenaga, K. Hirota

This study shows propofol causes metabolic reprogramming and cell death by impairing mitochondrial function, specifically complexes I and III, and this effect is exacerbated by preexisting mitochondrial dysfunction.

bioRxiv · Nov 16, 2017Pharmacology & Toxicology