Author

Weixi Feng

Weill Cornell MedicinePrimary ORCID profile (0000-0002-7201-5688 · 2 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Weixi FengiD2 papers · Nanjing Medical University

Biology Disciplines

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Neuroscience· 2 Systems Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:protein structure· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Jingjie ZhuiD· 3 shared Se-In Lee· 3 shared Haiyuan YuiD· 2 shared Katie Munechika· 2 shared Kendra Norman· 2 shared Li GaniD· 2 shared Man Ying WongiD· 2 shared Sadaf AminiD· 2 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Jingjie ZhuORCID

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Author profile →

3 papers

Recent·Most cited

TRAPPC8 Is an Endogenous Brake on UFMylation That Suppresses Tauopathy

Yuansong Wan, Ethan Cordes, Weixi Feng, Se-In Lee, Katie Munechika, Yu Sun, Zhisong Gao, Beichen Gao, Jingjie Zhu, Man Ying Wong, Kendra Norman, Siyu Wang, hao chen, Bangyan Liu, Zhong Li, , Li Gan

This study identifies TRAPPC8 as a brake on UFMylation that suppresses tauopathy by interacting with UBA5, showing its therapeutic potential in Alzheimer's disease.

bioRxiv · Aug 23, 2026 · PreprintNeuroscience

Interactome mapping in human excitatory neurons reveals novel risk genes and pathways in Alzheimer's disease

Xiaomu Wei, Katie Munechika, Yu Sun, Yuansong Wan, Tianyu Xia, Yuan Hou, Wenqiang Song, Kumar Yugandhar, Yiwen Wang, Se-In Lee, Zhengdong Sha, Yadi Zhou, Weixi Feng, Jingjie Zhu, Y. S. Tang, , Haiyuan Yu

The paper develops a neuron-specific interactome map in human excitatory neurons to identify novel risk genes and pathways in Alzheimer's disease.

bioRxiv · Mar 16, 2026 · PreprintSystems Biology

cGAS inhibition delays TDP-43-driven ALS Pathogenesis

Yajing Liu, Weixi Feng, Abulimiti Aikedan, Se-In Lee, Maitreyee Bhagwat, Ravi Kumar Nagiri, Man Ying Wong, Sadaf Amin, Wenhui Qu, Jingjie Zhu, Si-Yu Wang, Pearly Ye, Kendra Norman, Guillermo Coronas‐Samano, Marta Olah, , Li Gan

This study identifies cGAS as a key mediator of TDP-43 pathology in ALS and shows that its inhibition reduces RNA splicing defects and neurodegeneration.

bioRxiv · Feb 26, 2026 · PreprintNeuroscience