Author

Xuan Yu

Chinese Academy of Medical Sciences & Peking Union Medical College

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Approach:experimental assay· 1 Organism:human· 1 Assay:protein structure· 1

Frequent Co-Authors

Klaus‐Armin NaveiD· 2 shared Simone EggertiD· 2 shared Wiebke MöbiusiD· 2 shared Alons LendsiD· 1 shared Andrew Octavian SasmitaiD· 1 shared Anja Ronnenberg· 1 shared Anne-Fleur WildenburgiD· 1 shared Antoine LoquetiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersNeuroscience

Klaus‐Armin NaveORCID

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3 papers

Recent·Most cited

Transcriptional dynamics of the oligodendrocyte lineageand its regulation by the brain erythropoietin system

Liu Ye, Vinícius Daguano Gastaldi, Yasmina Curto, Anne-Fleur Wildenburg, Xuan Yu, Martin Hindermann, Simone Eggert, Anja Ronnenberg, Qing Wang, Umer Javed Butt, Riki Kawaguchi, Daniel H. Geschwind, Wiebke Möbius, Susann Boretius, Manvendra Singh, , Hannelore Ehrenreich

This study investigates how erythropoietin (EPO) and its receptor (EPOR) regulate oligodendrocyte lineage dynamics in the adult mouse hippocampus, showing EPO promotes differentiation and maturation, and suggests potential therapeutic applications for remyelination.

Nature Communications · Sep 16, 2025Peer-reviewedNeuroscience

Oligodendrocytes and neurons contribute to amyloid-β deposition in Alzheimer's disease

Andrew Octavian Sasmita, Constanze Depp, T. I. Nazarenko, Ting Sun, Sophie B. Siems, Erinne Cherisse Ong, Yakum Bertrand Nkeh, Carolin Böhler, Xuan Yu, Bastian Bues, Lisa Evangelista, Shuying Mao, Bárbara Morgado, Zoe Y. Wu, Torben Ruhwedel, , Klaus‐Armin Nave

The paper shows oligodendrocytes contribute to amyloid-β plaques in Alzheimer's disease, but neurons are required for plaque formation.

Nature Neuroscience · Aug 5, 2024Peer-reviewedNeuroscience

Emergence of stealth polymorphs that escape α-synuclein amyloid monitoring, take over and acutely spread in neurons

Francesca De Giorgi, Florent Laferrière, Federica Zinghirino, Emilie Faggiani, Alons Lends, Mathilde Bertoni, Xuan Yu, Axelle Grélard, Estelle Morvan, Birgit Habenstein, Nathalie Dutheil, Évelyne Doudnikoff, Jonathan Daniel, Stéphane Claverol, Chuan Qin, , François Ichas

Stealth α-synuclein fibrils escape ThT detection, outcompete visible polymorphs, and spread neurodegeneratively in vivo.

Science Advances · Oct 2, 2020Peer-reviewedNeuroscience