Author

Y. Cormerais

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

G. Pages· 2 shared I. Ben-Sahra· 2 shared S. Giuliano· 2 shared B. D. Manning· 1 shared C. M. Adams· 1 shared E. Tambutte· 1 shared G. Hoxhaj· 1 shared H. Endou· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

G. Pages

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Exploiting De Novo Serine Synthesis as a Metabolic Vulnerability to Overcome Sunitinib Resistance in Advanced Renal Cell Carcinoma

M. Teisseire, U. Sahu, J. Parola, M.-C. Tsai, V. Vial, J. Durivault, R. Grepin, Y. Cormerais, G. Pages, I. Ben-Sahra, S. Giuliano

This study identifies serine synthesis as a metabolic vulnerability in sunitinib-resistant renal cell carcinoma, showing that inhibiting key enzymes enhances drug sensitivity.

bioRxiv · Mar 22, 2024 · PreprintCancer Biology

Hepatic mTORC1 signaling activates ATF4 as part of its metabolic response to feeding and insulin

V. Byles, Y. Cormerais, K. Kalafut, V. Barrera, J. E. Hughes Hallett, S. Ho Sui, J. M. Asara, C. M. Adams, G. Hoxhaj, I. Ben-Sahra, B. D. Manning

This study shows that mTORC1 activates ATF4 in the liver to regulate amino acid metabolism during feeding and insulin signaling.

bioRxiv · May 2, 2021Cell Biology

Pharmacological inhibition of the amino-acid transporter LAT1 demonstrates in vitro anti-neoplastic activity towards medulloblastoma

Y. Cormerais, M. Pagnuzzi-Boncompagni, S. Schrötter, S. Giuliano, E. Tambutte, H. Endou, M. F. Wempe, G. Pages, J. Pouyssegur, V. Picco

This study shows that JPH203, a LAT1 inhibitor, effectively reduces medulloblastoma cell proliferation and survival without harming normal cells.

bioRxiv · Jan 8, 2019Cancer Biology