Author

Yining Zhao

Centre National de la Recherche ScientifiquePrimary ORCID profile (0009-0006-4828-5254 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Yining ZhaoiD1 paper · Washington University in St. Louis

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

Elodie Le Seac’h· 1 shared Elodie Vimont· 1 shared Étienne KornobisiD· 1 shared Florence LarrousiD· 1 shared Guilherme Dias de MeloiD· 1 shared Guillaume Drumont· 1 shared Hervé BourhyiD· 1 shared Isabelle TardieuxiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Elodie Le Seac’h

Mutual co-authors in cluster

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1 paper

Recent·Most cited

SARS-CoV-2 ORF9b exploits mitochondrial recruitment to TOMM70 for proteasomal protection and tunes inflammatory remodeling during lung infection

Yining Zhao, Lauriane Kergoat, Guilherme Dias de Melo, Florence Larrous, Guillaume Drumont, Juan Diego Hernández‐Camacho, Elodie Vimont, Elodie Le Seac’h, Nell Saunders, Martin Kächele, Étienne Kornobis, Quentin Giai Gianetto, Mariette Matondo, Isabelle Tardieux, Olivier Schwartz, , Timothy Wai

This study shows SARS-CoV-2 ORF9b uses TOMM70 to avoid proteasomal degradation, reduces inflammation, and is not essential for viral replication or lung pathology.

bioRxiv · Aug 3, 2026 · PreprintCell Biology