Joseph F. Cheer

This study shows that prenatal THC exposure and maternal stress impair maternal behavior and alter offspring neurodevelopment with sex- and region-specific molecular changes.

This study shows that prenatal THC exposure in male rats increases vulnerability to addiction-like behaviors by enhancing cue-driven dopamine responses and striatal encoding during reward seeking.

This study shows that CB1R signaling in the dorsomedial striatum affects Pavlovian outcome devaluation and inhibitory synaptic transmission, with sex differences observed in these effects.

The paper shows that two endocannabinoids, 2-AG and AEA, signal to different cell types (neurons and astrocytes), inducing distinct synaptic regulation mechanisms.

The paper develops a predictive model of cocaine relapse using dopamine release patterns and sex, showing that these factors predict relapse and extinction resistance.

This study shows that reducing CB1R signaling in the VTA decreases sign-tracking behavior and alters cue-outcome dopamine dynamics in the NAc of rats.

This study shows that prenatal THC exposure does not alter mesolimbic dopamine function or behavior in female rats, unlike males.

iAChSnFR, a fluorescent indicator built from a bacterial periplasmic binding protein, reports acetylcholine with large, fast signals in mouse, fish, fly and worm preparations.