Author
Allison W. Roberts
University of California, BerkeleyORCID 0000-0001-6681-4144Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2019-2026 · peak 2/yrCo-Author Collaboration Network
10 top collaborators · 17 mutual co-author links5 papers
Recent·Most cited
- BioengineeringPLoS ONEPeer-reviewed
Leveraging a synthetic biology approach to enhance BCG-mediated expansion of Vγ9Vδ2 T cells
Christine M. Qabar, Allison W. Roberts, Lucas M. Waldburger…·Jeffery S. Cox
This paper uses synthetic biology to engineer BCG to produce HMBPP, enhancing Vγ9Vδ2 T cell expansion without feedback inhibition, offering a strategy to improve TB vaccine efficacy.
2 citations - ImmunologybioRxivPreprint
Genome-wide screen in Mycobacterium tuberculosis infected macrophages reveals innate regulation of antibacterial mediators by IRF2
Allison W. Roberts, Linda N. Del Cid, Nicholas E. Garelis, Jeffery S. Cox
The paper identifies IRF2 as a regulator of antibacterial responses in Mtb-infected macrophages through a genome-wide CRISPR screen, showing its inhibitory role in macrophage activation.
3 citations - MicrobiologybioRxivPreprint
An unusual genetic switch controls Mycobacterium avium pathogenesis, antibiotic resistance and colony morphology
Hannah Nilsson, Zoe Netter, Miles I. Tuncel…·Jeffery S. Cox
This study identifies a genetic switch involving IS1245 transposition and Erp/MarP that controls colony morphology, virulence, and antibiotic resistance in Mycobacterium avium.
- MicrobiologybioRxivPreprint
Global proteomic profiling of primary macrophages during M. tuberculosis infection identifies TAX1BP1 as a mediator of autophagy targeting
Jonathan M. Budzik, Nick E. Garelis, Teresa Repasy…·Jeffery S. Cox
The paper uses proteomic analysis to identify TAX1BP1 as a mediator of autophagy targeting M. tuberculosis during macrophage infection.
DOI 10.1101/534917