Author

Yujia Liu

University of CambridgeORCID 0000-0002-0406-8569Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Yujia Liu1 paper · Lawrence Berkeley National Laboratory

Biology Disciplines

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Biophysics· 1 Neuroscience· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Organism:bacteria (other)· 1 Organism:C. elegans· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Aaron R. DinneriD· 1 shared Chris C. ChiiD· 1 shared Christian Schwall· 1 shared Dorota Skowronska‐KrawczykiD· 1 shared Genea EdwardsiD· 1 shared Gopal K. PattanayakiD· 1 shared Guy A PerkinsiD· 1 shared Helen Yoo· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperBiophysics

Aaron R. DinnerORCID

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3 papers

Recent·Most cited

Mixed positive and negative feedback loops drive diverse single-cell gene expression dynamics

Torkel E. Loman, Christian Schwall, Teresa Saez, Yujia Liu, James C W Locke

The paper uses stochastic modeling and experiments to show mixed feedback loops in bacteria generate diverse gene expression dynamics.

Nature Communications · Aug 17, 2026Peer-reviewedSystems Biology

An underlying bistability sets amplitude and explains temperature compensation in the cyanobacterial circadian clock

Yujia Liu, Gopal K. Pattanayak, Chris C. Chi, Helen Yoo, Aaron R. Dinner, Michael J. Rust

The paper identifies bistability in the cyanobacterial circadian clock as a mechanism for temperature compensation, linking it to amplitude regulation and phosphorylation dynamics.

bioRxiv · May 5, 2026 · PreprintBiophysics

Loss of AKAP1 triggers Drp1 dephosphorylation-mediated mitochondrial fission and loss in retinal ganglion cells

Genea Edwards, Guy A Perkins, Keun-young Kim A, YeEun Kong, Yonghoon Lee, Soo‐Ho Choi, Yujia Liu, Dorota Skowronska‐Krawczyk, Robert Neal Weinreb, Linda M. Zangwill, Stefan Strack, Won‐Kyu Ju

This study shows that loss of AKAP1 leads to Drp1 dephosphorylation, mitochondrial fragmentation, and RGC dysfunction in glaucoma, suggesting AKAP1 as a therapeutic target.

Cell Death and Disease · Apr 20, 2020Peer-reviewedNeuroscience