Author

Jessica N. Sowa

Baylor College of MedicineORCID 0000-0002-0896-7932Unclaimed ORCID profile · follow to get notified of new preprints

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Organism:C. elegans· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:E. coli· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Chih-Chun LiniD· 1 shared Christophe HermaniD· 1 shared David WangiD· 1 shared Efrem S. LimiD· 1 shared Emily R. TroemeliD· 1 shared Isaiah A.A. NeveiD· 1 shared Jay NiiD· 1 shared Johan PanekiD· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperGenetics

Chih-Chun LinORCID

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3 papers

Recent·Most cited

Novel infection by Mucor hiemalis kills Caenorhabditis hosts through intestinal perforation

Jay Ni, Spencer Weigand, Jessica N. Sowa

This study identifies Mucor hiemalis as a pathogen of Caenorhabditis nematodes, showing it kills hosts via intestinal perforation and does not trigger typical immune responses.

Infection and Immunity · Apr 15, 2026Peer-reviewedMicrobiology

Escherichia coli Metabolite Profiling Leads to the Development of an RNA Interference Strain for Caenorhabditis elegans

Isaiah A.A. Neve, Jessica N. Sowa, Chih-Chun Lin, Priya Sivaramakrishnan, Christophe Herman, Youqiong Ye, Leng Han, Meng Carla Wang

The paper identifies specific bacterial metabolites affecting C. elegans physiology and develops an RNA interference strain to study genotype-phenotype interactions without microbial confounding.

G3 Genes Genomes Genetics · Nov 11, 2019Peer-reviewedGenetics

An Intracellular Pathogen Response Pathway Promotes Proteostasis In C. elegans

Kirthi C. Reddy, Tal Dror, Jessica N. Sowa, Johan Panek, Kevin Chen, Efrem S. Lim, David Wang, Emily R. Troemel

This study identifies a novel pathway, the Intracellular Pathogen Response (IPR), that promotes proteostasis in C. elegans by upregulating ubiquitin ligase components like cul-6, with pals-22 acting as a repressor.

Current Biology · Nov 1, 2017Peer-reviewedGenetics