Author

Herbert Hildebrandt

Medizinische Hochschule HannoverORCID 0000-0002-1044-0881Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Alexandre StellaiD· 1 shared Alexei GorelikiD· 1 shared Alexey V. PshezhetskyiD· 1 shared Angelo PalmigianoiD· 1 shared Anja K. Münster-KühneliD· 1 shared Anne Gonzalez de PeredoiD· 1 shared Aurélien BoyanceiD· 1 shared Bénédicte AmilhoniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

Alexandre StellaORCID

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Author profile →

2 papers

Recent·Most cited

ST8SIA4-mediated polysialylation is critical for CCR2-driven monocyte egress from the bone marrow

Aurélien Boyance, Nelly Gilles, Maxime Rotival, Alexandre Stella, Camille Gorlt, Karima Chaoui, Sarah Monard, Nacho Aguiló, Thomas Mangeat, Odile Burlet‐Schiltz, Hugues Lortat‐Jacob, Anja K. Münster-Kühnel, Anne Gonzalez de Peredo, Denis Hudrisier, Lluís Quintana‐Murci, , Yoann Rombouts

This study identifies ST8SIA4-mediated polysialylation as essential for CCR2-driven monocyte egress from the bone marrow, revealing a novel regulatory mechanism in immune cell trafficking.

bioRxiv · Jan 12, 2026 · PreprintImmunology

Secondary deficiency of neuraminidase 1 contributes to CNS pathology in neurological mucopolysaccharidoses via hypersialylation of brain glycoproteins

TianMeng Xu, Rachel Heon‐Roberts, Travis Moore, Patricia Dubot, Xuefang Pan, Tianlin Guo, Christopher W. Cairo, Rebecca J. Holley, Brian Bigger, Thomas M. Durcan, Thierry Levade, Jérôme Ausseil, Bénédicte Amilhon, Alexei Gorelik, Bhushan Nagar, , Alexey V. Pshezhetsky

This study shows that reduced NEU1 activity in neurological MPS leads to hypersialylation of brain glycoproteins, contributing to CNS pathology through disrupted synaptogenesis and behavior.

Journal of Clinical Investigation · Jun 17, 2025Peer-reviewedBiochemistry