Julie A. Kauer

The paper shows that somatodendritic release of cholecystokinin from dopamine neurons in the VTA alters synaptic strength, both at GABAergic and glutamatergic synapses, and influences neighboring neurons.

This study uses CRISPR/Cas9 to knock down Clock in mouse VTA dopamine neurons, revealing effects on sleep, behavior, and excitability linked to bipolar disorder.

This study investigates the role of Hcrt receptor signaling in CRF neurons on alcohol drinking, anxiety, and BNST excitability, revealing sex-specific effects.

This study identifies the role of kappa opioid receptors in stress-induced cocaine seeking by showing their effect on GABAergic synapses in the ventral tegmental area.

This study identifies neuronal senescence in dorsal root ganglia of aged and injured mice, showing it contributes to sensory dysfunction and is reversible with senolytic treatment.

This study shows that loss of GPT2 in mice disrupts glutamate metabolism and synaptic transmission, with rescue by alpha-ketoglutarate supplementation.

This study shows that CCK released from somatodendritic regions of VTA dopamine neurons enhances GABAergic synapses and influences food consumption behaviors.

The paper introduces a method to control synaptically connected neurons using bioluminescent light, validated in cultured neurons and mice.

The paper investigates how NMDA receptor activation leads to long-term potentiation of glycinergic synapses in the spinal cord, potentially enhancing nociceptive signal transmission during injury.