Author

Jakob Maximilian Bader

Max Planck Institute of BiochemistryORCID 0000-0002-6575-0609Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Matthias ManniD· 2 shared Christian HaassiD· 1 shared Daniel Farny· 1 shared Dieter EdbaueriD· 1 shared Elena Katharina Schulz‐Trieglaff· 1 shared F. Ulrich HartliD· 1 shared Fabian HospiD· 1 shared Felix MeissneriD· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Matthias MannORCID

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2 papers

Recent·Most cited

Gel-like inclusions of C-terminal fragments of TDP-43 sequester and inhibit proteasomes in neurons

Henrick Riemenschneider, Qiang Guo, Jakob Maximilian Bader, Frédéric Frottin, Daniel Farny, Gernot Kleinberger, Christian Haass, Matthias Mann, F. Ulrich Hartl, Wolfgang P. Baumeister, Mark S. Hipp, Felix Meissner, Rubén Fernández‐Busnadiego, Dieter Edbauer

This study identifies TDP-25 inclusions as sequestering proteasomes, leading to proteostasis impairment in ALS/FTD.

bioRxiv · Mar 15, 2021 · PreprintNeuroscience

Cortical circuit alterations precede disease onset in Huntington’s disease mice

Johanna Burgold, Elena Katharina Schulz‐Trieglaff, Kerstin Voelkl, Sara Gutiérrez-Ángel, Jakob Maximilian Bader, Fabian Hosp, Matthias Mann, Thomas Arzberger, Rüdiger Klein, Sabine Liebscher, Irina Dudanova

This study shows that cortical circuit alterations, including increased neuronal activity and reduced inhibitory synapses, occur before disease onset in Huntington’s disease mice.

Scientific Reports · Apr 29, 2019Peer-reviewedNeuroscience