Author

Matthew L. Lawton

Boston UniversityORCID 0000-0002-7244-1337Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Developmental Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:benchmarking· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Alejandro Nico Rondón-OrtiziD· 1 shared Andrew CheniD· 1 shared Andrew EmiliiD· 1 shared Benjamin C. BlumiD· 1 shared Christian F. Heckendorf· 1 shared Christopher F. Thomas· 1 shared Cynthia A. BradhamiD· 1 shared Daniel T. ZuchiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Alejandro Nico Rondón-OrtizORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Plasma Exosomes in Insulin Resistant Obesity Exacerbate Progression of Triple Negative Breast Cancer

Pablo Llévenes, Andrew Chen, Matthew L. Lawton, Alejandro Nico Rondón-Ortiz, Yuhan Qiu, Michael Seen, Stefano Monti, Gerald V. Denis

This study shows that plasma exosomes from obese mice enhance TNBC aggressiveness by inducing epithelial-mesenchymal transition and Rac1 activation.

BMC Cancer · Jul 9, 2025Peer-reviewedCancer Biology

PANAMA enabled high sensitivity dual nanoflow LC/MS metabolomics and proteomics analysis

Weiwei Lin, Fatemeh Mousavi, Benjamin C. Blum, Christian F. Heckendorf, Matthew L. Lawton, Noah M. Lampl, Ryan Matthew Hekman, Hongbo Guo, Mark E. McComb, Andrew Emili

PANAMA enables high sensitivity joint metabolomics and proteomics analysis using nanoflow LC/MS without instrument duplication, improving resolution and detection sensitivity.

Cell Reports Methods · Jul 1, 2024Peer-reviewedSystems Biology

V-type H+ ATPase Activity is Required for Embryonic Dorsal-Ventral Symmetry Breaking

Daphne Schatzberg, Christopher F. Thomas, Patrick Reidy, Sarah E. Hadyniak, Viktoriya Skidanova, Matthew L. Lawton, Luz Dojer, Shweta Kitchloo, Daniel T. Zuch, Cynthia A. Bradham

This study shows that V-ATPase activity is required for DV symmetry breaking in sea urchin embryos, acting upstream of p38 MAPK and Nodal signaling.

bioRxiv · Oct 18, 2021 · PreprintDevelopmental Biology