Author

Yue Feng

Emory UniversityORCID 0000-0002-7905-2182Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Approach:experimental assay· 1

Frequent Co-Authors

Hongbing WangiD· 2 shared Qi DingiD· 2 shared Alex C. H. WongiD· 1 shared Bijay P. DhungeliD· 1 shared Bradley D. PearceiD· 1 shared Caroline GiardinaiD· 1 shared Carrie E. BeardeniD· 1 shared Charles G. BaileyiD· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersNeuroscience

Hongbing WangORCID

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Author profile →

4 papers

Recent·Most cited

Synaptic microRNA dysregulation orchestrates aberrant glutamatergic transmission in a human neuronal model of 22q11.2 deletion syndrome

Weibo Niu, Jidong Guo, Ximing Ran, David Parker, Xiangru Li, Shaojun Yu, Sidney Imes, Ryan H. Purcell, Christina Michalski, Eric Guo, Sophia Y Jiang, Ethan Xu, Gary Jonathan Bassell, Jennifer Gladys Mulle, Opal Ousley, , Zhexing Wen

This study identifies dysregulated synaptic microRNAs in 22q11DS neurons that drive aberrant glutamatergic transmission and synaptic plasticity, linking genetic deletion to psychosis risk.

bioRxiv · Oct 15, 2026 · PreprintNeuroscience

Intrinsically Disordered Regions Define Unique Protein Interaction Networks in CHD Family Remodelers

Mehdi Sharifi Tabar, Chirag Parsania, Caroline Giardina, Yue Feng, Alex C. H. Wong, Cynthia Metierre, Rajini Nagarajah, Bijay P. Dhungel, John E.J. Rasko, Charles G. Bailey

The paper identifies unique protein interaction networks in CHD family remodelers by analyzing intrinsically disordered regions and aggregation-prone regions, revealing their role in transcriptional regulation.

The FASEB Journal · May 15, 2025Peer-reviewedBiochemistry

Carbamazepine restores neuronal signaling, protein synthesis and cognitive function in a mouse model of fragile X syndrome

Qi Ding, Fan Zhang, Yue Feng, Hongbing Wang

This study shows that carbamazepine improves cognitive and behavioral deficits in a mouse model of fragile X syndrome by reducing ERK[1/2] and Akt signaling and protein synthesis.

International Journal of Molecular Sciences · Dec 7, 2020Peer-reviewedNeuroscience

Computational analysis of transcriptome signature repurposes low dose trifluoperazine for the treatment of fragile X syndrome in mouse model

Qi Ding, Ferzin Sethna, Xueting Wu, Zhuang Miao, Ping Chen, Yueqi Zhang, Hua Xiao, Wei Feng, Yue Feng, Xuan Li, Hongbing Wang

The paper identifies trifluoperazine as a potential treatment for FXS using transcriptome analysis and experimental validation.

Communications Biology · Mar 16, 2020Peer-reviewedNeuroscience