Author

Kendall M. Foote

Northwestern UniversityORCID 0000-0002-8086-5674Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

Geoffrey T. SwansoniD· 2 shared Anika P. WileniD· 1 shared Anis ContractoriD· 1 shared Brynna WebbiD· 1 shared Charlotte CastilloniD· 1 shared Emily A. BreachiD· 1 shared Erica Binelli· 1 shared Hieu TrinhiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Geoffrey T. SwansonORCID

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2 papers

Recent·Most cited

Wide-ranging behavioral dysfunction in two mouse models of pathological human variants in the GRIK2 kainate receptor gene

Brynna Webb, Hieu Trinh, Emily A. Breach, Kendall M. Foote, Erica Binelli, Geoffrey T. Swanson

This study creates mouse models of GRIK2 variants causing neurodevelopmental disorders and finds they exhibit behavioral and seizure-related impairments similar to humans.

bioRxiv · Aug 8, 2025 · PreprintNeuroscience

A pathogenic missense mutation in kainate receptors elevates dendritic excitability and synaptic integration through dysregulation of SK channels

Toshihiro Nomura, Sakiko Taniguchi, Yizhi Wang, Nai-Hsing Yeh, Anika P. Wilen, Charlotte Castillon, Kendall M. Foote, Jian Xu, John N. Armstrong, Jeffrey N. Savas, Geoffrey T. Swanson, Anis Contractor

This study investigates a missense mutation in the GRIK2 gene, showing it disrupts SK channel function, leading to increased dendritic excitability and synaptic integration in hippocampal neurons.

Journal of Neuroscience · Oct 6, 2023Peer-reviewedNeuroscience