Author

Talia E. Gileadi

Guy's HospitalORCID 0000-0002-8155-6822Unclaimed ORCID profile · follow to get notified of new preprints

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Neuroscience· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:protein structure· 1 Approach:statistical model· 1

Frequent Co-Authors

Angela CarusoiD· 1 shared Cathy FernandesiD· 1 shared Diana CashiD· 1 shared Eleanor HendyiD· 1 shared Jacob EllegoodiD· 1 shared Jason P. LerchiD· 1 shared Joyce Taylor‐Papadimitriou· 1 shared Karl-Peter GieseiD· 1 shared

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2024-2024 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Angela CarusoORCID

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1 paper

Recent·Most cited

Deficiency of the histone lysine demethylase KDM5B causes autism-like phenotypes via increased NMDAR signalling

Leticia Pérez‐Sisqués, S. Bhatt, Angela Caruso, Mohi U. Ahmed, Talia E. Gileadi, Shoshana Spring, Eleanor Hendy, Joyce Taylor‐Papadimitriou, Diana Cash, Nicholas E. Clifton, Jacob Ellegood, Laura C. Andreae, Jason P. Lerch, María Luisa Scattoni, Karl-Peter Giese, , M. Albert Basson

This study shows that KDM5B deficiency in mice leads to autism-like behaviors and increased brain size, which can be mitigated by NMDAR antagonists.

bioRxiv · May 28, 2024 · PreprintNeuroscience