Ulrich S. Schwarz

This study shows that geometric confinement and substrate stiffness influence cardiomyocyte force generation differently, with confinement enhancing contractile stress and relaxation on physiological substrates.

This study quantifies RBC shape recovery dynamics during Plasmodium falciparum infection, showing that parasite-induced membrane stiffening and knob architecture significantly affect relaxation time.

This study uses imaging to show how TRAP and CSP dynamics drive adhesion and trail formation in malaria sporozoites, revealing a link between adhesion and trail formation.

This study identifies a mechanism of asynchronous nuclear division in Plasmodium falciparum that enhances proliferation by minimizing resource idling through sequential allocation of a limiting replication resource.

This study shows how diatoms switch between one- and two-raphe contact gliding to modulate path curvature by dynamically altering cell-substrate contact sites.

This study shows that mechanosensing and IL-13 signaling work together through STAT6 and YAP to control intestinal stem cell differentiation and barrier function in fibrosis.

This paper compares volume and surface methods for microparticle traction force microscopy through simulations and experiments, finding the surface method more accurate but with reduced performance at high noise levels.

This study shows Lenacapavir causes HIV-1 capsid damage, exposing genomic DNA in nuclear speckles and altering capsid structure.

The paper models mechanochemical coupling in optogenetically activated cell layers using a finite element framework to understand signal propagation and cell responses.

This study uses traction force microscopy to show that TLP modulates force generation in malaria sporozoites during gliding motility.

This study uses 3D reconstructions and simulations to show that geometrical ratcheting stabilizes the shape of budding intestinal organoids through changes in tension and actomyosin reorganization.

This paper introduces DNA microbeads to control morphogen release in organoids, enabling spatio-temporal regulation of development and improving tissue mimicry.

This study identifies how IFITM3 inhibits influenza virus entry by stabilizing hemifusion through lipid sorting in endosomal membranes.

The paper shows clathrin-mediated endocytosis transitions from flat to curved structures when 70% of clathrin is assembled, driven by changes in clathrin-adaptor ratio and membrane tension.