Oliver Stork

This study investigates the effects of Bassoon gene ablation in specific mouse neuron populations on behavioral and structural phenotypes resembling schizophrenia.

This study shows that conditional ablation of the Bassoon gene in GABAergic interneurons leads to autism-like behaviors and synaptic dysfunction in mice.

This study uses a doxycycline-inducible 90CGG mouse model to show that FXAND features progress from hyperactivity to anxiety to motor dysfunction, with reversible electrophysiological and proteomic changes.

This study shows that increasing NPYergic transmission in the hippocampus of aged male mice rescues deficits in long-term potentiation in the dentate gyrus.

This study evaluates the therapeutic potential of trehalose in a mouse model of FXTAS, showing it reduces protein aggregation and improves motor function when administered preventively but not when used as a treatment.

The paper investigates sex differences in anxiety and threat avoidance in GAD65-/- mice, finding sex-specific changes in interneuron populations and neural oscillations in anxiety-related brain regions.

The paper investigates ECM remodeling in different epilepsy models and identifies ECM components that predict seizure properties.

This study shows that the dentate gyrus mediates stress-induced shifts in memory retrieval strategies in mice by regulating excitability through GAD65.

The paper shows that NPYergic circuitry in the dorsal dentate gyrus regulates remote fear memory generalization in mice through engram cell activity and neurotransmission recovery.

This study shows that antibiotic treatment in mice reduces synaptic transmission and cholinergic gamma oscillations in the hippocampus, linked to microglia activation and immune changes.

This study shows that synaptic activity controls DNA methylation by degrading DNMT3a1 via NMDAR-dependent neddylation.

This study shows SIPA1L2 connects TrkB-containing amphisomes to dynein for retrograde trafficking and local signaling at presynaptic terminals, essential for BDNF-dependent plasticity.