Author

Elise Quadri

Institute for Research in Immunology and CancerORCID 0009-0000-7875-0328Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

Aaron David SchimmeriD· 1 shared Abdula MaheriD· 1 shared Alex C.H. LiuiD· 1 shared Andrea ArrudaiD· 1 shared Anie MonastiD· 1 shared Ann-Sophie GironneiD· 1 shared Christopher B. MarshalliD· 1 shared Daina Zofija AvizonisiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

Aaron David SchimmerORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 12, 2026

CD59 organizes the plasma membrane to sustain oncogenic Ras-MAPK signaling and is a targetable vulnerability in acute myeloid leukemia

Abdula Maher, Dhanoop Manikoth Ayyathan, Séverine Cathelin…·Steven M. Chan

This study identifies CD59 as a critical regulator of Ras-MAPK signaling in AML, showing its depletion impairs leukemic growth and identifies rILYd4 as a potential therapeutic target.

DOI 10.64898/2026.08.11.744285

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintSep 4, 2025

EZH2 Inhibition Induces a Metabolic Stress Response Sensitizing TNBC to Glutaminase Targeting

Lucas Porras, Marina Fukano, Ann-Sophie Gironne…·Geneviève Deblois

This study shows that EZH2 inhibition in TNBC causes metabolic stress, making cells more sensitive to glutaminase inhibitors.

DOI 10.1101/2025.09.04.674332

humanproteomics

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