Author

Marisa Di Monaco

University of DundeeORCID 0009-0004-1293-9266Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Alain J. Kemp· 1 shared Alessandro VasconiD· 1 shared Alex von KriegsheimiD· 1 shared Carla Salomo-ColliD· 1 shared Christian SchellhaasiD· 1 shared Damian J. MoleiD· 1 shared Gabriella SaksonoiD· 1 shared Hywel Dunn-DaviesiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Alain J. Kemp

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJul 21, 2026

Epithelial-Mesenchymal Transition Induces FAM134A-mediated ER-phagy to Regulate Procollagen Secretion

Marisa Di Monaco, Özlem Şen, Gabriella Saksono…·Jin Rui Liang

The paper shows that EMT activates ER-phagy to regulate procollagen secretion, with FAM134A uniquely mediating this process through a non-canonical mechanism.

DOI 10.64898/2026.07.20.739610

humanproteomics

𝕏 PostBluesky

Cancer BiologyDevelopmental CellPeer-reviewedAug 15, 2025

Dysproteostasis primes pancreatic epithelial state changes in KRAS-mediated oncogenesis

Carla Salomo-Coll, Marisa Di Monaco, Jocelyn Holkham…·Simon Wilkinson

This study shows that proteostatic failure in ER-phagy, driven by Kras, leads to protein aggregates that prime acinar-ductal metaplasia and pancreatic cancer.

7 citationstop 4%

mouseproteomics

𝕏 PostBluesky