Science Translational MedicineMolecular BiologyPeer-reviewed journal article
A cell-penetrant peptide blocking C9ORF72-repeat RNA nuclear export suppresses neurodegeneration
Lydia M. CastelliiD, Ya-Hui LiniD, Álvaro Sánchez-MartíneziD, Aytaç GüliD, Kamallia Mohd ImraniD, Adrian Higginbottom, Santosh Kumar UpadhyayiD, Nóra M. MárkusiD, Raquel Rua MartinsiD, Johnathan Cooper‐KnockiD, Claire MontmassoniD, Rebecca N. Cohen, Amy Walton, Claudia S. Bauer, Kurt J. De VosiD, Richard J. MeadiD, Mimoun AzzouziD, Cyril DomingueziD, Laura FerraiuoloiD, Pamela J. ShawiD, Alexander J. WhitworthiD, Guillaume M. HautbergueiD
Abstract
Hexanucleotide repeat expansions in C9ORF72 are the most common genetic cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD), a spectrum of incurable debilitating neurodegenerative diseases. Here, we report a novel ALS/FTD drug concept with in vivo and in vitro therapeutic activity in preclinical models of C9ORF72-ALS/FTD. Our data demonstrate that supplementation or oral administration of a cell-penetrant peptide, which competes with the SRSF1:NXF1 interaction, confers neuroprotection by inhibiting the nuclear export of pathological C9ORF72-repeat transcripts in various models of disease including primary neurons, patient-derived motor neurons and Drosophila. Our drug-like rationale for disrupting the nuclear export of microsatellite repeat transcripts in neurological disorders provides a promising alternative to conventional small molecule inhibitors often limited by poor blood-brain barrier penetrance.
- organism: human
- organism: Drosophila
- research focus: drug response / drug discovery
- approach: experimental assay
- Citations
- 16
- FWCI
- 2.54
- Field percentile
- top 10%
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