Author

A. H. Baker

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Bradshaw· 1 shared A. Kurkiewicz· 1 shared A. Lemckert· 1 shared A. Molinolo· 1 shared A. Shaw· 1 shared B. A. Scheven· 1 shared C. Delles· 1 shared C. van der Zalm· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

A. Bradshaw

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Human AdV-20-42-42, a promising novel adenoviral vector for gene therapy and vaccine product development

M. Z. Ballmann, S. Raus, R. Engelhart, G. L. Kajan, C. van der Zalm, T. Papp, L. John, S. Khan, J. Custers, W. A. M. Bakker, H. M. van der Schaar, N. Arnberg, A. Lemckert, M. Havenga, A. H. Baker

This paper describes the development of a novel adenoviral vector, HAdV-20-42-42, which shows promise for gene therapy and vaccination due to its low seroprevalence and effective transduction capabilities.

bioRxiv · Mar 11, 2021Immunology

The common analgesic paracetamol enhances the anti-tumour activity of decitabine through exacerbation of oxidative stress

H. J. Gleneadie, A. H. Baker, N. Batis, J. Bryant, Y. Jiang, S. J. Clokie, H. Mehanna, P. Garcia, D. M. Gendoo, S. Roberts, A. Molinolo, S. Gutkind, B. A. Scheven, P. R. Cooper, F. Khanim, M. Wiench

This study shows that paracetamol enhances decitabine's anti-tumour effects by increasing oxidative stress without worsening DNA damage.

bioRxiv · Mar 31, 2020 · PreprintCancer Biology

Transforming growth factor-beta signaling via ALK1 and ALK5 regulates distinct functional pathways in vein graft intimal hyperplasia

A. Bradshaw, E. Low, J. T. Schwartze, A. Kurkiewicz, M. Pek, D. Kelly, A. Shaw, M. Thorikay, J. McClure, M. McBride, S. Arias-Rivas, S. E. Francis, N. Morrell, C. Delles, P. Herzyk, , A. H. Baker

This study shows TGF{beta} signals through ALK1 and ALK5 in vein graft SMCs, with ALK5 promoting contractility and ALK1 driving migration, suggesting balancing these pathways could prevent graft failure.

bioRxiv · Nov 29, 2019 · PreprintCell Biology