Author

Dipa Bhaumik

Buck Institute for Research on Aging

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Systems Biology· 2 Cell Biology· 1

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Research Focus:differential expression· 3 Organism:C. elegans· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Anna FoulgeriD· 3 shared Birgit SchillingiD· 3 shared Christina D. KingiD· 3 shared Gordon J. LithgowiD· 3 shared Julie Kay AnderseniD· 3 shared Edward AndertoniD· 2 shared Manish ChamoliiD· 2 shared Angelina HolcomiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersSystems Biology

Anna FoulgerORCID

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3 papers

Recent·Most cited

Shifts in protein aggregate stability define proteostasis decline in the aging human brain

Edward Anderton, Jordan B. Burton, Christina D. King, Anna Foulger, Dipa Bhaumik, Daria Timonina, Zachary Mayeri, Manish Chamoli, Julie Kay Andersen, Birgit Schilling, Gordon J. Lithgow

This study shows that human brain aging involves asymmetric remodeling of the insoluble proteome, with intermediate-stability aggregates accumulating and being linked to neurodegenerative disease.

bioRxiv · Mar 30, 2026 · PreprintCell Biology

Amyloid β accelerates age-related proteome-wide protein insolubility.

Edward Anderton, Manish Chamoli, Dipa Bhaumik, Christina D. King, Xueshu Xie, Anna Foulger, Julie Kay Andersen, Birgit Schilling, Gordon J. Lithgow

This study shows that amyloid β accelerates age-related protein insolubility in C. elegans and links this process to neurodegenerative and age-related diseases through proteomics and GWAS.

GeroScience · May 16, 2024Peer-reviewedSystems Biology

Neuronal expression of human amyloid-β and Tau drives global phenotypic and multi-omic changes in C. elegans

Angelina Holcom, Matías Fuentealba, Renuka Sivapatham, Christina D. King, Hadley Osman, Anna Foulger, Dipa Bhaumik, Birgit Schilling, David Furman, Julie Kay Andersen, Gordon J. Lithgow

This study shows that expressing human amyloid-β and Tau in C. elegans neurons causes phenotypic and multi-omic changes, including accelerated aging and metabolic dysfunction.

bioRxiv · Jun 1, 2023 · PreprintSystems Biology