Author

G. Veit

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:protein folding & dynamics· 2 Assay:proteomics· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Assay:cryo-EM· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

G. L. Lukacs· 3 shared H. Xu· 3 shared F. Jiang· 2 shared J. C. Kappes· 2 shared Z. Yang· 2 shared A. Bidaud-Meynard· 1 shared A. Mezzell· 1 shared A. Premchandar· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersBiophysics

G. L. Lukacs

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Author profile →

3 papers

Recent·Most cited

Folding correctors can restore CFTR posttranslational folding landscape by allosteric domain-domain coupling

G. L. Lukacs, N. Soya, H. Xu, A. Roldan, Z. Yang, H. Ye, A. Premchandar, F. Jiang, G. Veit, S. P. C. Cole, J. C. Kappes, T. Hegedus

The paper shows that allosteric domain-domain coupling modulates the folding landscape of ABCC transporters, and that folding correctors can rescue misfolding by rewiring these networks.

bioRxiv · Oct 19, 2023Biophysics

Mechanism of dual pharmacological correction and potentiation of human CFTR

C. Wang, Z. Yang, B. J. Loughlin, H. Xu, G. Veit, S. Vorobiev, O. B. Clarke, F. Jiang, Y. Li, S. Singh, Z. Rich, E. R. Menten, R. A. Grassucci, W. Wang, A. Mezzell, , J. F. Hunt

This paper uses cryo-EM to reveal how VX445 corrects CFTR folding and gating defects by altering allosteric communication pathways.

bioRxiv · Oct 10, 2022 · PreprintBiophysics

Transcytosis maintains CFTR apical polarity in the face of constitutive and mutation-induced basolateral missorting

A. Bidaud-Meynard, F. Bossard, A. Schnur, R. Fukuda, G. Veit, H. Xu, G. L. Lukacs

The paper shows that transcytosis helps maintain CFTR apical polarity by retrieving basolateral CFTR and counteracting missorting caused by mutations or loss of NHERF1 interaction.

bioRxiv · Oct 20, 2018Cell Biology