Author

J. M. Rosenbluth

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:organoid / primary culture· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

D. A. Dillon· 2 shared F. Lynce· 2 shared J. Z. Yu· 2 shared A. J. Graham· 1 shared B. Y. Nabet· 1 shared C. Biellak· 1 shared C. D. Morley· 1 shared E. J. Kim· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

D. A. Dillon

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Stress relaxing granular bioprinting materials enable complex and uniform organoid self-organization

A. J. Graham, M. W. L. Khoo, V. Srivastava, S. Viragova, H. Kim, K. Parekh, K. M. Hennick, M. Bird, N. Goldhammer, J. Z. Yu, G. Hu, N. T. Brinkley, L. Pardo, J. S. Amaya, C. D. Morley, , Z. J. Gartner

This paper introduces a tunable granular biomaterial, MAGIC matrix, that enables precise control of initial conditions for tissue growth, leading to improved organoid reproducibility and complexity.

bioRxiv · Feb 1, 2024Bioengineering

Implantation of engineered adipocytes that outcompete tumors for resources suppresses cancer progression

H. P. Nguyen, R. Sheng, E. Murray, Y. Ito, M. Bruck, C. Biellak, K. An, F. Lynce, D. A. Dillon, M. J. M. Magbanua, L. A. Huppert, H. Hammerlindl, L. Esserman, J. M. Rosenbluth, N. Ahituv

The paper shows engineered adipocytes can suppress cancer progression by competing for nutrients in multiple cancer types.

bioRxiv · Mar 28, 2023 · PreprintCancer Biology

T cells Instruct Immune Checkpoint Inhibitor Therapy Resistance in Tumors Responsive to IL-1 and TNFa Inflammation.

N. W. Cho, S. M. Guldberg, B. Y. Nabet, J. Z. Yu, E. J. Kim, K. J. Hiam-Galvez, J. L. Yee, R. DeBarge, I. Tenvooren, N. A. Ashitey, F. Lynce, D. A. Dillon, J. M. Rosenbluth, M. H. Spitzer

The paper identifies a novel mechanism of ICI resistance where T cells drive inflammatory resistance via IL-1 and TNF in tumors, with implications for therapy.

bioRxiv · Sep 20, 2022Immunology