Author

K. Blackwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Z. Wu· 2 shared A. J. M. Walhout· 1 shared F. Zhang· 1 shared F. Zhu· 1 shared H. Na· 1 shared H.-Y. Qu· 1 shared J. Campos· 1 shared J. Ferreira· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

Z. Wu

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Mild impairment of mitochondrial function increases longevity and pathogen resistance through ATFS-1-driven activation of p38-regulated innate immunity

J. Campos, Z. Wu, P. D. Rudich, S. Soo, M. Mistry, J. Ferreira, K. Blackwell, J. Van Raamsdonk

This paper shows that mild mitochondrial dysfunction in C. elegans extends lifespan and enhances pathogen resistance via ATFS-1-driven activation of p38-regulated innate immunity.

bioRxiv · Mar 31, 2021Cell Biology

ERK signaling licenses SKN-1A/NRF1 for proteasome production and proteasomal stress resistance

P. Zhang, H.-Y. Qu, Z. Wu, H. Na, J. Hourihan, F. Zhang, F. Zhu, M. Isik, A. J. M. Walhout, Y.-X. Feng, K. Blackwell

This study identifies ERK1/2-mediated phosphorylation of NRF1 as critical for proteasome production and stress resistance, showing therapeutic potential in cancer.

bioRxiv · Jan 4, 2021 · PreprintCell Biology