Author

Katharina Overhoff

Max Planck Institute for Multidisciplinary Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Andrew Octavian SasmitaiD· 2 shared Constanze DeppiD· 2 shared Gesine SaheriD· 2 shared Klaus‐Armin NaveiD· 2 shared Lena SpiethiD· 2 shared Oliver WirthsiD· 2 shared Stefan A. BerghoffiD· 2 shared Swati SubramanianiD· 2 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Andrew Octavian SasmitaORCID

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3 papers

Recent·Most cited

A genus-wide interaction atlas across NS4B orthologues identifies a conserved role for UFMylation in orthoflavivirus replication

Sreejith Rajasekharan, Viviana Andrea Barragan Torres, Yago Côrtes Pinheiro Gomes, Lucas Wilken, Katharina Overhoff, Yen-Chia Lin, Shunmoogum A. Patten, Satoru Watanabe, Laurent Chatel‐Chaix, Pietro Scaturro

This study identifies conserved interactions of NS4B with the UFMylation pathway across orthoflaviviruses, revealing a novel role for UFMylation in viral replication and suggesting it as a therapeutic target.

Nature Communications · Mar 13, 2026Peer-reviewedMicrobiology

Oligodendrocytes and neurons contribute to amyloid-β deposition in Alzheimer's disease

Andrew Octavian Sasmita, Constanze Depp, T. I. Nazarenko, Ting Sun, Sophie B. Siems, Erinne Cherisse Ong, Yakum Bertrand Nkeh, Carolin Böhler, Xuan Yu, Bastian Bues, Lisa Evangelista, Shuying Mao, Bárbara Morgado, Zoe Y. Wu, Torben Ruhwedel, , Klaus‐Armin Nave

The paper shows oligodendrocytes contribute to amyloid-β plaques in Alzheimer's disease, but neurons are required for plaque formation.

Nature Neuroscience · Aug 5, 2024Peer-reviewedNeuroscience

Ageing-associated myelin dysfunction drives amyloid deposition in mouse models of Alzheimer's disease

Constanze Depp, Ting Sun, Andrew Octavian Sasmita, Lena Spieth, Stefan A. Berghoff, T. I. Nazarenko, Katharina Overhoff, Agnes A. Steixner-Kumar, Swati Subramanian, Sahab Arinrad, Torben Ruhwedel, Wiebke Möbius, Sandra Göbbels, Gesine Saher, Hauke B. Werner, , Klaus‐Armin Nave

This study shows that ageing-related myelin dysfunction drives amyloid-beta deposition in Alzheimer's disease mouse models, suggesting myelin integrity as an upstream risk factor.

Nature · May 31, 2023Peer-reviewedNeuroscience