Author

Kristin L. McNally

National Institutes of Health

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:metagenomics· 1 Organism:mouse· 1

Frequent Co-Authors

Sonja M. BestiD· 3 shared Adam HageiD· 2 shared Aaron CarmodyiD· 1 shared Abhilash I. Chiramel· 1 shared Angela L. RasmusseniD· 1 shared Brady N. ZelliD· 1 shared Byron ShueiD· 1 shared Charles L. LarsoniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersMicrobiology

Sonja M. BestORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Epigenetic regulation of DPP4 receptor expression by NONO enables replication of MERS-CoV

Adam Hage, Mikhaila Janes, Seth D. Scott, Suhyeon Yoon, Kevin Rose, Tovah E. Markowitz, Lara M. Myers, Aaron Carmody, Lenny Triem, Olivia Straw, Kristin L. McNally, Paul A. Beare, Justin Lack, Craig Martens, Sonja M. Best

This study identifies NONO as a host factor that epigenetically regulates DPP4 expression, facilitating MERS-CoV replication by modulating histone modifications at the DPP4 locus.

bioRxiv · Sep 23, 2026 · PreprintMicrobiology

Restraint of Powassan virus replication by TRIM5α facilitates viral avoidance of antiviral immunity

Rebecca M. Broeckel, Emily A. Fitzmeyer, Eva Chebishev, Jeffrey G. Shannon, Byron Shue, Adam Hage, Stephanie J. Spada, Efrosini Artikis, Abhilash I. Chiramel, Kristin L. McNally, Charles L. Larson, Maarit von Kutzleben, Shelly J. Robertson, Danielle K. Offerdahl, Hilary Ann Lakin, , Sonja M. Best

The paper investigates how TRIM5α restricts orthoflavivirus replication and identifies a mutation in POWV's NS3 gene that allows it to evade TRIM5α inhibition.

Communications Biology · Jul 7, 2026Peer-reviewedMicrobiology

Novel Mechanism of Inflammatory Activation by Ebola Virus Matrix Protein Linked to the Ebolavirus Virulence

Satoko Yamaoka, Zeineb Mhamdi, Lin Wang, Vaille A. Swenson, Kristin L. McNally, Shao‐Chia Lu, Reema Singh, Stephanie L. Saundh, Brady N. Zell, Sonja M. Best, Michael A. Barry, Angela L. Rasmussen, Hideki Ebihara

This study identifies VP40 as a key virulence factor in Ebola virus that drives pro-inflammatory responses through NF-κB activation in non-immune cells, independent of TLR4.

Proceedings of the National Academy of Sciences · Dec 29, 2025Peer-reviewedMicrobiology