Author

Ling Zhu

The University of Sydney

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Neuroscience· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:clustering· 1

Frequent Co-Authors

Aakriti Bhandari· 1 shared Andrew Chang· 1 shared Chi ZhangiD· 1 shared Christa L. MontgomeryiD· 1 shared Elisa Eleanor CornishiD· 1 shared Felicia A. Juarez· 1 shared Haeyn Lim· 1 shared Jennifer D. BallheimiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Aakriti Bhandari

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

IOP-induced blood-retinal barrier compromise contributes to RGC death in glaucoma

Chi Zhang, Haeyn Lim, Jennifer D. Ballheim, Marina Simón, Rui Fu, Nicholas G. Tolman, Logan J. Horbal, Felicia A. Juarez, Qing Wang, Aakriti Bhandari, Christa L. Montgomery, Ling Zhu, Jonathan Guy Crowston, Nicolas Robine, Michael P. Fautsch, , Simon W. M. John

This study shows that elevated intraocular pressure compromises the blood-retinal barrier, leading to vascular leakage and retinal ganglion cell death in glaucoma models.

Nature Communications · Apr 8, 2026Peer-reviewedNeuroscience

Spatial multi-omics unveils the monoclonal origin, neuroendocrine plasticity, and microenvironment niches in combined small cell lung cancer

Zhuo Wang, Qing Luo, Jie Wu, Li Lu, Wenjie Ding, Yichun Zhao, Yongfeng YU, Ruoran Qiu, Ling Zhu, Xinxing Ouyang, Wendi Xuzhang, Shun Lu, Wei Wei, Qihui Shi, Ziming Li

This study uses spatial multi-omics to show cSCLC has a monoclonal origin, lineage plasticity, and distinct microenvironment niches, leading to a new diagnostic assay.

Cell Reports Medicine · Apr 1, 2026Peer-reviewedCancer Biology

Dysregulated Proline Metabolism Contributes to Subretinal Fibrosis in Neovascular AMD: Therapeutic Potential of Prolyl-4-Hydroxylase Inhibition

Yue Zeng, Ting Zhang, Elisa Eleanor Cornish, Sora Lee, Michelle Yam, Mark Eminhizer, Jingwen Zeng, Jialing Zhang, Shaoxue Zeng, Xian Wei, Jinliang Yang, Meidong Zhu, Andrew Chang, Meixia Zhang, Jianhai Du, , Mark C. Gillies

This study identifies proline metabolism, specifically proline hydroxylation via P4HA1, as a driver of subretinal fibrosis in nAMD and shows that P4HA1 inhibition reduces fibrosis in mouse models.

bioRxiv · Apr 13, 2025 · PreprintNeuroscience