Author

M. Hollinshead

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:electronic health records· 1

Frequent Co-Authors

A. Cavazza· 1 shared A. Clements· 1 shared A. J. Thrasher· 1 shared A. Worth· 1 shared C. Hess· 1 shared E. Rivers· 1 shared G. Elliott· 1 shared G. L. Smith· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperPhysiology

A. Cavazza

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Novel role for ESCRT-III component CHMP4C in the integrity of the endocytic network utilized for herpes simplex virus envelopment

T. Russell, J. Samolej, M. Hollinshead, G. L. Smith, J. Kite, G. Elliott

The paper identifies CHMP4C as a critical fission factor required for HSV1 envelopment by maintaining the integrity of the recycling endocytic network.

bioRxiv · Aug 19, 2020Cell Biology

Wiskott Aldrich syndrome protein regulates non-selective autophagy and mitochondrial homeostasis in human myeloid cells

E. Rivers, R. Rai, J. Lotscher, M. Hollinshead, G. Markelj, J. Thaventheran, A. Worth, A. Cavazza, C. Hess, M. Bajaj-Elliott, A. J. Thrasher

This study shows that WASp deficiency impairs autophagy and mitochondrial function in human myeloid cells, with ARP2/3 dependency and metabolic consequences.

bioRxiv · Feb 6, 2020Physiology

Shigella sonnei infection of zebrafish reveals that O-antigen mediates neutrophil tolerance and dysentery incidence

V. Torraca, M. Kaforou, J. Watson, G. M. Duggan, H. Guerrero-Gutierrez, S. Krokowski, M. Hollinshead, T. B. Clarke, R. J. Mostowy, G. S. Tomlinson, V. Sancho-Shimizu, A. Clements, S. Mostowy

This paper shows that Shigella sonnei's O-antigen mediates neutrophil tolerance and increases dysentery incidence in zebrafish, suggesting O-antigen as a therapeutic target.

PLOS Pathogens · Dec 12, 2019Peer-reviewedMicrobiology