Author

M. Schafer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Lagnado· 1 shared A. P. Bluhm· 1 shared A. Parini· 1 shared A. Sobh· 1 shared A. Tagmount· 1 shared A. Walaszczyk· 1 shared C. Correia-Melo· 1 shared C. D. Vulpe· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. Lagnado

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Genome-scale CRISPR Screens Identify Host Factors that Promote Human Coronavirus Infection

M. Grodzki, A. P. Bluhm, M. Schafer, A. Tagmount, M. Russo, A. Sobh, R. Rafiee, C. D. Vulpe, S. M. Karst, M. H. Norris

Genome-wide CRISPR screens identified host factors that promote human coronavirus infection, revealing novel antiviral drug targets.

bioRxiv · Jun 4, 2021Microbiology

Length-independent telomere damage drives cardiomyocyte senescence

R. Anderson, A. Lagnado, D. Maggiorani, A. Walaszczyk, E. Dookun, J. Chapman, J. Birch, H. Salmonowicz, M. Ogrodnik, D. Jurk, C. Proctor, C. Correia-Melo, S. Victorelli, E. Fielder, R. Berlinguer-Palmini, , J. F. Passos

This study shows that telomere damage independent of length causes cardiomyocyte senescence, leading to cardiac dysfunction, and that clearing senescent cells improves cardiac health.

bioRxiv · Aug 17, 2018Cell Biology