Author

Martin Lothar Biniossek

University of Freiburg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1

Frequent Co-Authors

Sheikh NizamuddiniD· 2 shared Stefanie Koidl· 2 shared Adrianna Seredynska· 1 shared Andrea PrunottoiD· 1 shared Andrea Ritter· 1 shared Armelle ArnouxiD· 1 shared Bettina A. Mayer· 1 shared Bettina Liebowitz Knapp· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersMolecular Biology

Sheikh NizamuddinORCID

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Author profile →

3 papers

Recent·Most cited

TAF2 condensation in nuclear speckles links basal transcription factor TFIID to RNA splicing

Tanja Bhuiyan, Niccolò Arecco, Paulina Karen Mendoza Sanchez, Juhyeong Kim, Carsten Schwan, Sophie Weyrauch, Sheikh Nizamuddin, Andrea Prunotto, Mehmet Tekman, Martin Lothar Biniossek, Bettina Liebowitz Knapp, Stefanie Koidl, Friedel Drepper, Pitter F. Huesgen, Robert Grosse, , Sebastian J. Arnold

This study shows TAF2's intrinsically disordered region drives condensation in nuclear speckles, recruiting SRRM2 to TFIID and affecting alternative splicing.

Cell Reports · Apr 25, 2025Peer-reviewedMolecular Biology

Serum Proteome Profiling Identifies N-Cadherin and C-Met as Early Marker Candidates of Therapeutic Response to Neoadjuvant Chemotherapy in Breast Cancer

Ines Derya Steenbuck, Miguel Cosenza‐Contreras, Klemens Fröhlich, Bettina A. Mayer, Konrad Kurowski, Tilman Werner, Meike Reinold, Matthias Fahrner, Frank Hause, Adrianna Seredynska, Tobias Feilen, Andrea Ritter, Armelle Arnoux, Martin Lothar Biniossek, Daniela Weiß, , Oliver Schilling

bioRxiv · May 24, 2024 · PreprintCancer Biology

Multi-omics analyses of MEN1 missense mutations identify disruption of menin-MLL and menin-JunD interactions as critical requirements for molecular pathogenicity

Koen M.A. Dreijerink, Ezgi Ozyerli‐Goknar, Stefanie Koidl, Ewoud J. van der Lelij, Priscilla van den Heuvel, Jeffrey J. Kooijman, Martin Lothar Biniossek, Kees W. Rodenburg, Sheikh Nizamuddin, H. T. Marc Timmers

The paper identifies MEN1 missense mutations that disrupt menin-MLL and menin-JunD interactions, highlighting their role in pathogenicity.

Epigenetics & Chromatin · Aug 8, 2022Peer-reviewedMolecular Biology