Author

Martina Cerisoli

University of Zurich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Adriano AguzziiD· 3 shared Davide CaredioiD· 2 shared Elena De CeccoiD· 2 shared Giovanni MariuttiiD· 2 shared Lukas D. FrickiD· 2 shared Andrea ArmaniiD· 1 shared Anna Maria ReussiD· 1 shared Beatrice GattaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersPathology

Adriano AguzziORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

aDISCO: A clearing method to enable 3D microscopyof large archival paraffin-embedded human tissue blocks

Anna Maria Reuss, Dominik Groos, Martina Cerisoli, Lena Nordberg, Lukas D. Frick, Fabian F. Voigt, Nikita Vladimirov, Philipp Bethge, Regina Reimann, Fritjof Helmchen, Peter Rupprecht, Adriano Aguzzi

aDISCO enables 3D microscopy of large archival FFPE human tissue blocks, revealing subtle pathologies in focal cortical dysplasia through deep-learning analysis.

Science Advances · Aug 12, 2026Peer-reviewedPathology

Prion propagation is controlled by a hierarchical network involving the nuclear Tfap2c and hnRNP K factors and the cytosolic mTORC1 complex

Stefano Sellitto, Davide Caredio, Matteo Bimbati, Giovanni Mariutti, Martina Cerisoli, Lukas D. Frick, Vangelis Bouris, Carlos Omar Oueslati Morales, Dalila Laura Vena, Sandesh Neupane, Federico Baroni, Kathi Ging, Jiang‐An Yin, Elena De Cecco, Andrea Armani, Adriano Aguzzi

The paper identifies TFAP2C as a genetic interactor of hnRNP K, showing their roles in mTOR metabolic regulation and a causal link to prion propagation.

PLoS Pathogens · Apr 20, 2026Peer-reviewedCell Biology

Gpnmb Defines a Phagocytic State of Microglia Linked to Neuronal Loss in Prion Disease

Davide Caredio, Giovanni Mariutti, Lisa K. Polzer, Martina Cerisoli, Beatrice Gatta, Yasmine Laimeche, Giulia Miracca, Marc Emmenegger, Marián Hruška-Plocháň, Magdalini Polymenidou, Matthias Schmitz, Inga Zerr, Elena De Cecco, Adriano Aguzzi

The paper identifies Gpnmb as a marker of a phagocytic microglial state linked to neuronal loss in prion disease, showing its role in clearing apoptotic cells.

bioRxiv · Feb 11, 2025 · PreprintNeuroscience